Nuclear targeting of a midregion PTHrP fragment is necessary for stimulating growth in breast cancer cells.
about
Lessons from in vitro studies and a related intracellular angiotensin II transgenic mouse model.Absence of PTHrP nuclear localization and carboxyl terminus sequences leads to abnormal brain development and function.Nuclear trafficking of secreted factors and cell-surface receptors: new pathways to regulate cell proliferation and differentiation, and involvement in cancers.PTHrP produced by myeloma plasma cells regulates their survival and pro-osteoclast activity for bone disease progression.PTHrP inhibits BMP-6 expression through the PKA signaling pathway in breast cancer cells.Endothelin-1 (ET-1) increases the expression of remodeling genes in vascular smooth muscle through linked calcium and cAMP pathways: role of a phospholipase A(2)(cPLA(2))/cyclooxygenase-2 (COX-2)/prostacyclin receptor-dependent autocrine loop.Deficiency of the parathyroid hormone-related peptide nuclear localization and carboxyl terminal sequences leads to premature skin ageing partially mediated by the upregulation of p27.
P2860
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P2860
Nuclear targeting of a midregion PTHrP fragment is necessary for stimulating growth in breast cancer cells.
description
2006 nî lūn-bûn
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2006年の論文
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2006年論文
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2006年論文
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2006年論文
@zh-hk
2006年論文
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2006年論文
@zh-tw
2006年论文
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2006年论文
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2006年论文
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name
Nuclear targeting of a midregi ...... growth in breast cancer cells.
@en
type
label
Nuclear targeting of a midregi ...... growth in breast cancer cells.
@en
prefLabel
Nuclear targeting of a midregi ...... growth in breast cancer cells.
@en
P2093
P2860
P356
P1476
Nuclear targeting of a midregi ...... growth in breast cancer cells.
@en
P2093
John F Robertson
Rajendra Kumari
Sue A Watson
P2860
P356
10.1002/IJC.21802
P577
2006-07-01T00:00:00Z