Increased CTLA-4+ T cells may contribute to impaired T helper type 1 immune responses in patients with chronic obstructive pulmonary disease.

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Increased CTLA-4+ T cells may contribute to impaired T helper type 1 immune responses in patients with chronic obstructive pulmonary disease.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

@zh-hk

2017年論文

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2017年論文

@zh-tw

2017年论文

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2017年论文

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2017年论文

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Increased CTLA-4+ T cells may ...... obstructive pulmonary disease.

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label

Increased CTLA-4+ T cells may ...... obstructive pulmonary disease.

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prefLabel

Increased CTLA-4+ T cells may ...... obstructive pulmonary disease.

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P1476

Increased CTLA-4+ T cells may ...... obstructive pulmonary disease

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P2093

Abdul M Setiawan

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Maja Zimmermann

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Nathanael E Ong

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Patricia Price

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Teck-Hui Teo

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Yuben P Moodley

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P2860

Global Strategy for the Diagnosis, Management, and Prevention of Chronic Obstructive Pulmonary Disease

Haemophilus influenzae oral vaccination for preventing acute exacerbations of chronic bronchitis and chronic obstructive pulmonary disease

CTLA-4 and PD-1 receptors inhibit T-cell activation by distinct mechanisms

CTLA-4 blockade in tumor models: an overview of preclinical and translational research

Global strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease: GOLD executive summary

Current understanding of macrophage type 1 cytokine responses during intracellular infections.

Investigation of soluble and transmembrane CTLA-4 isoforms in serum and microvesicles.

Interleukin-6 induces early gamma interferon production in the infected lung but is not required for generation of specific immunity to Mycobacterium tuberculosis infection

Treg/IL-17 ratio and Treg differentiation in patients with COPD.

Single dose of anti-CTLA-4 enhances CD8+ T-cell memory formation, function, and maintenance

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219-226

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scholarly article

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10.1111/IMM.12725

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P433

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P478

151

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Dino Tan

Lea-Ann Kirkham

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2017-03-20T00:00:00Z

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P698

28190271

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P921

chronic obstructive pulmonary disease

P932

5418458

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