Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
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Glyoxalase 1 increases anxiety by reducing GABAA receptor agonist methylglyoxalSpatial correlation between brain aerobic glycolysis and amyloid-β (Aβ ) depositionAPP processing in Alzheimer's diseaseGender-Specific Hippocampal Dysrhythmia and Aberrant Hippocampal and Cortical Excitability in the APPswePS1dE9 Model of Alzheimer's DiseaseKv3.4 is modulated by HIF-1α to protect SH-SY5Y cells against oxidative stress-induced neural cell deathCholesterol enhances classical conditioning of the rabbit heart rate responseMitochondrial Ca2+ overload underlies Abeta oligomers neurotoxicity providing an unexpected mechanism of neuroprotection by NSAIDsPotassium 2-(1-hydroxypentyl)-benzoate promotes long-term potentiation in Aβ1-42-injected rats and APP/PS1 transgenic mice.Alzheimer's proteins, oxidative stress, and mitochondrial dysfunction interplay in a neuronal model of Alzheimer's disease.Notoginsenoside R1 increases neuronal excitability and ameliorates synaptic and memory dysfunction following amyloid elevation.Voltage-gated potassium channels at the crossroads of neuronal function, ischemic tolerance, and neurodegeneration.High dietary cholesterol facilitates classical conditioning of the rabbit's nictitating membrane response.Systemic administration of substance P recovers beta amyloid-induced cognitive deficits in rat: involvement of Kv potassium channels.Preventing effect of L-type calcium channel blockade on electrophysiological alterations in dentate gyrus granule cells induced by entorhinal amyloid pathologyChanges in the physiology of CA1 hippocampal pyramidal neurons in preplaque CRND8 mice.Linking aβ42-induced hyperexcitability to neurodegeneration, learning and motor deficits, and a shorter lifespan in an Alzheimer's modelBACE1 gene deletion prevents neuron loss and memory deficits in 5XFAD APP/PS1 transgenic miceProgressive effect of beta amyloid peptides accumulation on CA1 pyramidal neurons: a model study suggesting possible treatmentsLinking vascular disorders and Alzheimer's disease: potential involvement of BACE1Aberrant dendritic excitability: a common pathophysiology in CNS disorders affecting memory?The Alzheimer's disease beta-secretase enzyme, BACE1.Neuritin activates insulin receptor pathway to up-regulate Kv4.2-mediated transient outward K+ current in rat cerebellar granule neurons.Physiological roles for amyloid beta peptides.Neuregulin-1/ErbB4 signaling regulates Kv4.2-mediated transient outward K+ current through the Akt/mTOR pathwayThe Basic Biology of BACE1: A Key Therapeutic Target for Alzheimer's Disease.Prion protein and Alzheimer disease.Pore formation by human stefin B in its native and oligomeric states and the consequent amyloid induced toxicity.Effects of ginsenoside Rg1 on the expression of toll-like receptor 3, 4 and their signalling transduction factors in the NG108-15 murine neuroglial cell line.Amyloid β inhibits retinoic acid synthesis exacerbating Alzheimer disease pathology which can be attenuated by an retinoic acid receptor α agonistAmyloid-beta1-42 reduces neuronal excitability in mouse dentate gyrus.Differential expression of voltage-gated K+ currents in medial septum/diagonal band complex neurons exhibiting distinct firing phenotypes.Activation of G-protein-gated inwardly rectifying potassium (Kir3/GirK) channels rescues hippocampal functions in a mouse model of early amyloid-β pathology.Age-related decrease in stimulated glutamate release and vesicular glutamate transporters in APP/PS1 transgenic and wild-type mice.Impact of amyloid-beta changes on cognitive outcomes in Alzheimer's disease: analysis of clinical trials using a quantitative systems pharmacology model.
P2860
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P2860
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en-gb
type
label
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en-gb
prefLabel
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en-gb
P2093
P1476
Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current.
@en
P2093
Darragh B Freir
Hugh A Pearson
Leigh D Plant
Martin Ramsden
Nicola J Webster
P304
P356
10.1016/J.NEUROBIOLAGING.2005.09.038
P577
2005-11-04T00:00:00Z