Infection of human intestinal epithelial cells by invasive bacteria activates NF-κB and increases ICAM-1 expression through NOD1.

about

Expression and in vitro assessment of tumorigenicity for NOD1 and NOD2 receptors in breast cancer cell lines.

P2860

Q55314093-D341FBD9-40CC-46B2-A775-8FA3FC3FE231

P2860

Infection of human intestinal epithelial cells by invasive bacteria activates NF-κB and increases ICAM-1 expression through NOD1.

Item

http://www.wikidata.org/entity/Q40370130

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Infection of human intestinal ...... CAM-1 expression through NOD1.

@en

type

Item

label

Infection of human intestinal ...... CAM-1 expression through NOD1.

@en

prefLabel

Infection of human intestinal ...... CAM-1 expression through NOD1.

@en

P1476

Infection of human intestinal ...... CAM-1 expression through NOD1.

@en

P2093

Bum Joon Park

http://www.w3.org/2001/XMLSchema#string

Jae Gyu Kim

http://www.w3.org/2001/XMLSchema#string

Sung Joong Lee

http://www.w3.org/2001/XMLSchema#string

Woon Geon Shin

http://www.w3.org/2001/XMLSchema#string

P2860

Cell differentiation is a key determinant of cathelicidin LL-37/human cationic antimicrobial protein 18 expression by human colon epithelium

Nod1 is an essential signal transducer in intestinal epithelial cells infected with bacteria that avoid recognition by toll-like receptors

The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5

Production of MDC/CCL22 by human intestinal epithelial cells

Decreased expression of Toll-like receptor-4 and MD-2 correlates with intestinal epithelial cell protection against dysregulated proinflammatory gene expression in response to bacterial lipopolysaccharide

Leukocyte adhesion deficiency syndromes: adhesion and tethering defects involving beta 2 integrins and selectin ligands

Signalling pathways and molecular interactions of NOD1 and NOD2

Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract

Nod1 detects a unique muropeptide from gram-negative bacterial peptidoglycan

Human intestinal epithelial cells are broadly unresponsive to Toll-like receptor 2-dependent bacterial ligands: implications for host-microbial interactions in the gut.

P304

81-90

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3904/KJIM.2015.409

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-01-16T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28092699

http://www.w3.org/2001/XMLSchema#string

P921

NF-κB

P932

5768537

http://www.w3.org/2001/XMLSchema#string

Infection of human intestinal epithelial cells by invasive bacteria activates NF-κB and increases ICAM-1 expression through NOD1.

about

P2860

P2860

Infection of human intestinal epithelial cells by invasive bacteria activates NF-κB and increases ICAM-1 expression through NOD1.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698

P921

P932