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Repeated mild traumatic brain injury: mechanisms of cerebral vulnerability.

Repeated mild traumatic brain injury: mechanisms of cerebral vulnerability.

http://www.wikidata.org/entity/Q40385671

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Treatments for traumatic brain injury with emphasis on transcranial near-infrared laser phototherapy Repetitive concussions in adolescent athletes - translating clinical and experimental research into perspectives on rehabilitation strategies Animal models of sports-related head injury: bridging the gap between pre-clinical research and clinical reality Concussion under-reporting and pressure from coaches, teammates, fans, and parents.Investigating the properties of the hemodynamic response function after mild traumatic brain injury Found in translation: Understanding the biology and behavior of experimental traumatic brain injury The acute phase of mild traumatic brain injury is characterized by a distance-dependent neuronal hypoactivity.Blast Overpressure Waves Induce Transient Anxiety and Regional Changes in Cerebral Glucose Metabolism and Delayed Hyperarousal in Rats A novel closed-head model of mild traumatic brain injury caused by primary overpressure blast to the cranium produces sustained emotional deficits in mice.Neuroimaging in repetitive brain trauma.Chronic cerebrovascular dysfunction after traumatic brain injury Considerations for Experimental Animal Models of Concussion, Traumatic Brain Injury, and Chronic Traumatic Encephalopathy-These Matters Matter.Examining the psychometric properties of a sport-related concussion survey: a Rasch measurement approach Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.Neuropsychological and neurobehavioral outcome following childhood arterial ischemic stroke: attention deficits, emotional dysregulation, and executive dysfunction.The collective therapeutic potential of cerebral ketone metabolism in traumatic brain injury An introduction to sports concussions.The pathophysiology underlying repetitive mild traumatic brain injury in a novel mouse model of chronic traumatic encephalopathy.Genetic activation of mTORC1 signaling worsens neurocognitive outcome after traumatic brain injury.Metabolic Response of Pediatric Traumatic Brain Injury.Functional Neuroimaging Distinguishes Posttraumatic Stress Disorder from Traumatic Brain Injury in Focused and Large Community Datasets.The Temporal Pattern of Changes in Serum Biomarker Levels Reveals Complex and Dynamically Changing Pathologies after Exposure to a Single Low-Intensity Blast in Mice The new neurometabolic cascade of concussion.Resonance of human brain under head acceleration.Enteral Nutrition for Patients With Traumatic Brain Injury in the Rehabilitation Setting: Associations With Patient Preinjury and Injury Characteristics and Outcomes.Repeated Closed Head Injury in Mice Results in Sustained Motor and Memory Deficits and Chronic Cellular Changes.Frequency of head-impact-related outcomes by position in NCAA division I collegiate football players.Concussion Education in U.S. Collegiate Sport: What Is Happening and What Do Athletes Want?Concussion management in US college football: progress and pitfalls.Additional Post-Concussion Impact Exposure May Affect Recovery in Adolescent Athletes.Elucidating the severity of preclinical traumatic brain injury models: a role for functional assessment?Behavioral, blood, and magnetic resonance imaging biomarkers of experimental mild traumatic brain injury.A novel head-neck cooling device for concussion injury in contact sports.GFAP-BDP as an acute diagnostic marker in traumatic brain injury: results from the prospective transforming research and clinical knowledge in traumatic brain injury study.Emergence of cognitive deficits after mild traumatic brain injury due to hyperthermia.Therapeutic targeting of the axonal and microvascular change associated with repetitive mild traumatic brain injury.Microglial neuroinflammation contributes to tau accumulation in chronic traumatic encephalopathy.Influences of developmental age on the resolution of diffuse traumatic intracranial hemorrhage and axonal injury.Frequent mild head injury promotes trigeminal sensitivity concomitant with microglial proliferation, astrocytosis, and increased neuropeptide levels in the trigeminal pain system.Evidence for the therapeutic efficacy of either mild hypothermia or oxygen radical scavengers after repetitive mild traumatic brain injury.

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P2860

Repeated mild traumatic brain injury: mechanisms of cerebral vulnerability.

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Repeated mild traumatic brain injury: mechanisms of cerebral vulnerability.

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Christopher C Giza

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David A Hovda

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Daya Alexander

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Linking adolescent sleep, brain maturation, and behavior

The adolescent brain and age-related behavioral manifestations

Cerebrovascular reactivity to CO(2) and hypotension after mild cortical impact injury.

Mapping cerebral glucose metabolism during spatial learning: interactions of development and traumatic brain injury.

The [14C]deoxyglucose method for the measurement of local cerebral glucose utilization: theory, procedure, and normal values in the conscious and anesthetized albino rat.

Pediatric sports-related concussion produces cerebral blood flow alterations.

Metabolic recovery following human traumatic brain injury based on FDG-PET: time course and relationship to neurological disability.

Changes in cortical extracellular levels of energy-related metabolites and amino acids following concussive brain injury in rats.

Repeat traumatic brain injury in the juvenile rat is associated with increased axonal injury and cognitive impairments

Adolescent brain development and animal models.

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traumatic brain injury

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