about
P185
Caspase-8 prevents sustained activation of NF-kappaB in monocytes undergoing macrophagic differentiationSpecific involvement of caspases in the differentiation of monocytes into macrophagesInduction of transglutaminase 2 by a liver X receptor/retinoic acid receptor alpha pathway increases the clearance of apoptotic cells by human macrophagesFOXP3 expression in cancer cells and anthracyclines efficacy in patients with primary breast cancer treated with adjuvant chemotherapy in the phase III UNICANCER-PACS 01 trial.Bleomycin exerts ambivalent antitumor immune effect by triggering both immunogenic cell death and proliferation of regulatory T cellsBcl-2 proteins: targets and tools for chemosensitisation of tumor cells.Membrane-associated Hsp72 from tumor-derived exosomes mediates STAT3-dependent immunosuppressive function of mouse and human myeloid-derived suppressor cellsLiver X receptor β activation induces pyroptosis of human and murine colon cancer cellsIdentification of biological markers of liver X receptor (LXR) activation at the cell surface of human monocytes.T-bet expression in intratumoral lymphoid structures after neoadjuvant trastuzumab plus docetaxel for HER2-overexpressing breast carcinoma predicts survivalPhenolic extract from oleaster (Olea europaea var. Sylvestris) leaves reduces colon cancer growth and induces caspase-dependent apoptosis in colon cancer cells via the mitochondrial apoptotic pathway.Liver X receptor ligand cytotoxicity in colon cancer cells and not in normal colon epithelial cells depends on LXRβ subcellular localization.Does bevacizumab impact anti-EGFR therapy efficacy in metastatic colorectal cancer?Induction of pyroptosis in colon cancer cells by LXRβProduction of adenosine by ectonucleotidases: a key factor in tumor immunoescape.STAT3 activation: A key factor in tumor immunoescape.Sirtuin-1 Activation Controls Tumor Growth by Impeding Th17 Differentiation via STAT3 Deacetylation.Cytotoxic effects of chemotherapy on cancer and immune cells: how can it be modulated to generate novel therapeutic strategies?The transcription factor IRF1 dictates the IL-21-dependent anticancer functions of TH9 cells.SOCS3 transactivation by PPARγ prevents IL-17-driven cancer growth.Chemotherapy-triggered cathepsin B release in myeloid-derived suppressor cells activates the Nlrp3 inflammasome and promotes tumor growth.Dacarbazine-mediated upregulation of NKG2D ligands on tumor cells activates NK and CD8 T cells and restrains melanoma growth.5-Fluorouracil selectively kills tumor-associated myeloid-derived suppressor cells resulting in enhanced T cell-dependent antitumor immunity.7beta-Hydroxycholesterol and 25-hydroxycholesterol-induced interleukin-8 secretion involves a calcium-dependent activation of c-fos via the ERK1/2 signaling pathway in THP-1 cells: oxysterols-induced IL-8 secretion is calcium-dependent.Identification of proteins cleaved downstream of caspase activation in monocytes undergoing macrophage differentiation.Redistribution of CD95, DR4 and DR5 in rafts accounts for the synergistic toxicity of resveratrol and death receptor ligands in colon carcinoma cells.Translocation of the inhibitor of apoptosis protein c-IAP1 from the nucleus to the Golgi in hematopoietic cells undergoing differentiation: a nuclear export signal-mediated event.An atypical caspase-independent death pathway for an immunogenic cancer cell line.
P50
Q24307493-8DC17E01-D7C7-4055-B0B0-4C98BDC22D63Q24312229-BBB47547-DE88-4975-BB7A-D0A0CA1BE38FQ24317052-D4F96EBB-3521-44DB-A54B-5311A46BF019Q27851740-C45876F2-C7A6-47F9-8398-B796EFF2F95EQ28533749-BFC2F7D2-9FE9-4063-9E97-0EF0BAD01899Q33348190-6A92B759-1EEC-4EEE-9B05-AA086A4AADFBQ33604925-4384FF09-DDE9-4C33-8525-CD3941C1ADA1Q34486432-26A06A1B-132C-41CF-B828-37D1B73FE948Q34489752-0427F7EF-BCC4-45ED-AE89-3575F7611EE5Q35212682-E9D008A1-9DFE-4506-BC23-ED37CB7F264CQ36284206-4A43A636-FDC5-4CB2-AD5C-89EB46A96736Q36413574-DD8BB0A0-5E75-4E89-B187-E6F349FDC0C0Q36962594-3C398527-7778-4D0C-A3FC-3B4A5B356BEEQ36997797-3B7441AC-9ACA-4114-878A-555BE89F92BAQ38058320-6140BE91-5321-44A4-BF8F-0658399D6A4DQ38140901-F90D56D1-E501-4A63-A4D3-D9D78E7AF74CQ38818429-C57AD23C-BFBD-4A11-BF1D-CF769CDF5C59Q38963228-4C68986F-E8E6-47D6-B42B-5089390A3F38Q38980079-A4477B36-2D7D-4D08-B0EA-28BCD849461BQ39160299-DF7C44D4-27B5-419C-8785-B51C036C9641Q39234393-444A78B0-EE5C-48DB-88BE-0CA4849370EEQ39284974-9CA87096-FB9A-482B-985C-E4D1AC5409BEQ39715226-95C3AEEC-4539-4B15-8FED-AD1CD28F6102Q40006045-53A50CB1-81A9-4181-8C9A-F2A0F9DA91AFQ40288296-B6633D63-EE4C-413B-9CAB-3868AB1832DEQ40504942-64FB4B4D-B102-414D-8F63-08BE919F349AQ40547569-BC2E3B1D-019B-42E0-B9EC-13C8445D6C26Q40707613-298F374A-F563-4512-9E29-5729E6027D79
P50
description
onderzoeker
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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Cédric Rébé
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P214
P269
P106
P184
P2038
Cedric_Rebe
P21
P213
0000 0003 5780 8521
P214
P269
P31
P569
1976-01-01T00:00:00Z
P7859
viaf-199990602