Two time windows of anisomycin-induced amnesia for inhibitory avoidance training in rats: protection from amnesia by pretraining but not pre-exposure to the task apparatus.
about
Linking new information to a reactivated memory requires consolidation and not reconsolidation mechanismsTranscription factors in long-term memory and synaptic plasticityBDNF-triggered events in the rat hippocampus are required for both short- and long-term memory formationBDNF activates mTOR to regulate GluR1 expression required for memory formationChemogenetic inactivation of ventral hippocampal glutamatergic neurons disrupts consolidation of contextual fear memory.The formation of auditory fear memory requires the synthesis of protein and mRNA in the auditory thalamus.Memory consolidation for the discrimination of frequency-modulated tones in mongolian gerbils is sensitive to protein-synthesis inhibitors applied to the auditory cortex.Cognitive dysfunction is sustained after rescue therapy in experimental cerebral malaria, and is reduced by additive antioxidant therapy.Social recognition memory requires two stages of protein synthesis in mice.Retrieval of memory for fear-motivated training initiates extinction requiring protein synthesis in the rat hippocampus.Short- and long-term memory: differential involvement of neurotransmitter systems and signal transduction cascades.Effects of post-training hippocampal injections of midazolam on fear conditioning.Amnesia of inhibitory avoidance by scopolamine is overcome by previous open-field exposure.NMDA receptor subunits in the adult rat hippocampus undergo similar changes after 5 minutes in an open field and after LTP induction.CREB antisense oligodeoxynucleotide administration into the dorsal hippocampal CA3 region impairs long- but not short-term spatial memory in mice.A new device for step-down inhibitory avoidance task--effects of low and high frequency in a novel device for passive inhibitory avoidance task that avoids bioimpedance variationsLearning-induced arg 3.1/arc mRNA expression in the mouse brainThe extracellular protease matrix metalloproteinase-9 is activated by inhibitory avoidance learning and required for long-term memory.The role of protein synthesis during the labile phases of memory: revisiting the skepticism.Memory retrieval of inhibitory avoidance requires histamine H1 receptor activation in the hippocampusIntrahippocampal infusions of anisomycin produce amnesia: contribution of increased release of norepinephrine, dopamine, and acetylcholine.Negative Allosteric Modulation of mGluR5 Partially Corrects Pathophysiology in a Mouse Model of Rett Syndrome.A phosphodiesterase 4-controlled switch between memory extinction and strengthening in the hippocampus.Treatment of fear memories: interactions between extinction and reconsolidation.The regulation of transcription in memory consolidationTemporal phases of long-term potentiation (LTP): myth or fact?Late Protein Synthesis-Dependent Phases in CTA Long-Term Memory: BDNF Requirement.Fear Memory.Retrieval does not induce reconsolidation of inhibitory avoidance memory.Role of hippocampal signaling pathways in long-term memory formation of a nonassociative learning task in the ratTime-dependent changes in the mouse hippocampal synaptic membrane proteome after contextual fear conditioning.Anisomycin infused into the hippocampus fails to block "reconsolidation" but impairs extinction: the role of re-exposure duration.Il1-β involvement in cognitive impairment after sepsis.Acute brain inflammation and oxidative damage are related to long-term cognitive deficits and markers of neurodegeneration in sepsis-survivor rats.Differential involvement of cortical muscarinic and NMDA receptors in short- and long-term taste aversion memory.Dose-related effects of propericiazine in rats.Requirement of beta-adrenergic receptor activation and protein synthesis for LTP-reinforcement by novelty in rat dentate gyrus.Walnut supplementation reverses the scopolamine-induced memory impairment by restoration of cholinergic function via mitigating oxidative stress in rats: a potential therapeutic intervention for age related neurodegenerative disorders.Antioxidant treatment prevents cognitive impairment and oxidative damage in pneumococcal meningitis survivor rats.Inhibition of protein synthesis or mTOR in the basolateral amygdala blocks retrieval-induced memory strengthening.
P2860
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P2860
Two time windows of anisomycin-induced amnesia for inhibitory avoidance training in rats: protection from amnesia by pretraining but not pre-exposure to the task apparatus.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Two time windows of anisomycin ...... xposure to the task apparatus.
@en
type
label
Two time windows of anisomycin ...... xposure to the task apparatus.
@en
prefLabel
Two time windows of anisomycin ...... xposure to the task apparatus.
@en
P2093
P2860
P356
P1433
P1476
Two time windows of anisomycin ...... exposure to the task apparatus
@en
P2093
P2860
P304
P356
10.1101/LM.6.6.600
P577
1999-11-01T00:00:00Z