Hypoxic tumor-derived microvesicles negatively regulate NK cell function by a mechanism involving TGF-β and miR23a transfer. - Wikidata - wikimedia - TriplyDB

Hypoxic tumor-derived microvesicles negatively regulate NK cell function by a mechanism involving TGF-β and miR23a transfer.

Hypoxic tumor-derived microvesicles negatively regulate NK cell function by a mechanism involving TGF-β and miR23a transfer.

http://www.wikidata.org/entity/Q40437768

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Aberrant Lipid Metabolism Promotes Prostate Cancer: Role in Cell Survival under Hypoxia and Extracellular Vesicles Biogenesis Essential role of miRNAs in orchestrating the biology of the tumor microenvironment Transferring intercellular signals and traits between cancer cells: extracellular vesicles as "homing pigeons"Tumor-Derived Exosomes and Their Role in Tumor-Induced Immune Suppression Functional Roles for Exosomal MicroRNAs in the Tumour Microenvironment Exosomes in tumor microenvironment: novel transporters and biomarkers Hijacker of the Antitumor Immune Response: Autophagy Is Showing Its Worst Facet Deciphering the role of ectosomes in cancer development and progression: focus on the proteome.Current status and future prospects of peptide-based cancer vaccines.Understanding the epigenetic regulation of tumours and their microenvironments: opportunities and problems for epigenetic therapy.Cancer-derived exosomic microRNAs shape the immune system within the tumor microenvironment: State of the art.Functions of Cancer-Derived Extracellular Vesicles in Immunosuppression.Natural Killer Cells in the Orchestration of Chronic Inflammatory Diseases Tumor microenvironment and noncoding RNAs as co-drivers of epithelial-mesenchymal transition and cancer metastasis.NK Cell Exhaustion.Hepatocyte-derived exosomes promote T follicular regulatory cell expansion during HCV infection.Horizontal transfer of miR-23a from hypoxic tumor cell colonies can induce angiogenesis.NK-92 cell, another ideal carrier for chimeric antigen receptor.HIF-1-mediated production of exosomes during hypoxia is protective in renal tubular cells.Exosome-Based Cell-Cell Communication in the Tumor Microenvironment.The Double Face of Exosome-Carried MicroRNAs in Cancer Immunomodulation.The Origin and Functions of Exosomes in Cancer.Hypoxia and acidosis: immune suppressors and therapeutic targets.The Potential Biomarkers and Immunological Effects of Tumor-Derived Exosomes in Lung Cancer.Genotoxic stress modulates the release of exosomes from multiple myeloma cells capable of activating NK cell cytokine production: Role of HSP70/TLR2/NF-kB axis.Cancer-derived extracellular vesicles: friend and foe of tumour immunosurveillance.Immune evasion mediated by PD-L1 on glioblastoma-derived extracellular vesicles.Effect of colorectal cancer-derived extracellular vesicles on the immunophenotype and cytokine secretion profile of monocytes and macrophages.MicroRNA meta-signature of oral cancer: evidence from a meta-analysis.MiR-23a transcriptional activated by Runx2 increases metastatic potential of mouse hepatoma cell via directly targeting Mgat3.Hypoxia, Metabolism and Immune Cell Function.Role of hypoxia-induced exosomes in tumor biology Effect of exosomal miRNA on cancer biology and clinical applications Sensitive and rapid quantification of exosomes by fusing luciferase to exosome marker proteins Exosome-mediated regulation of tumor immunology MicroRNAs: Pleiotropic Regulators in the Tumor Microenvironment

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Hypoxic tumor-derived microvesicles negatively regulate NK cell function by a mechanism involving TGF-β and miR23a transfer.

http://www.wikidata.org/entity/Q40437768

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

@zh-sg

2015年學術文章

@yue

2015年學術文章

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2015年學術文章

@zh-hant

name

Hypoxic tumor-derived microves ...... ing TGF-β and miR23a transfer.

@en

type

label

Hypoxic tumor-derived microves ...... ing TGF-β and miR23a transfer.

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prefLabel

Hypoxic tumor-derived microves ...... ing TGF-β and miR23a transfer.

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2162402X.2015.1062968

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Hypoxic tumor-derived microves ...... ing TGF-β and miR23a transfer.

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Arash Nanbakhsh

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Bassam Janji

http://www.w3.org/2001/XMLSchema#string

Etienne Moussay

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Fathia Mami-Chouaib

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Manon Bosseler

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Muhammad Zaeem Noman

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Salem Chouaib

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Sonia Baconnais

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P2860

Microvesicles: mediators of extracellular communication during cancer progression

Innate and adaptive immune cells in the tumor microenvironment

Tumor-derived microvesicles: shedding light on novel microenvironment modulators and prospective cancer biomarkers

Exosomes reflect the hypoxic status of glioma cells and mediate hypoxia-dependent activation of vascular cells during tumor development

Functions of natural killer cells

Hypoxic enhancement of exosome release by breast cancer cells.

Granzyme B degradation by autophagy decreases tumor cell susceptibility to natural killer-mediated lysis under hypoxia.

PD-L1 is a novel direct target of HIF-1α, and its blockade under hypoxia enhanced MDSC-mediated T cell activation.

Tumor-derived exosomes are a source of shared tumor rejection antigens for CTL cross-priming.

Hypoxia-inducible factors and RAB22A mediate formation of microvesicles that stimulate breast cancer invasion and metastasis.

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Jerome Paggetti

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27141372

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