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Myofibroblast contraction activates latent TGF-beta1 from the extracellular matrixThe myofibroblast: one function, multiple originsThe myofibroblast in wound healing and fibrosis: answered and unanswered questionsRecent developments in myofibroblast biology: paradigms for connective tissue remodelingThe role of the myofibroblast in tumor stroma remodelingMyofibroblasts and mechano-regulation of connective tissue remodellingIsoform-specific regulation of the actin-organizing protein palladin during TGF-beta1-induced myofibroblast differentiationIntegrins and the activation of latent transforming growth factor beta1 - an intimate relationshipAlpha-smooth muscle actin is crucial for focal adhesion maturation in myofibroblasts.Focal adhesion size controls tension-dependent recruitment of alpha-smooth muscle actin to stress fibersPrestress in the extracellular matrix sensitizes latent TGF-β1 for activation.Integrins Form an Expanding Diffusional Barrier that Coordinates Phagocytosis.Plasma fibronectin stabilizes Borrelia burgdorferi-endothelial interactions under vascular shear stress by a catch-bond mechanismIntegrins αvβ5 and αvβ3 promote latent TGF-β1 activation by human cardiac fibroblast contractionWound-healing defect of CD18(-/-) mice due to a decrease in TGF-beta1 and myofibroblast differentiation.The single-molecule mechanics of the latent TGF-β1 complex.Cells lacking β-actin are genetically reprogrammed and maintain conditional migratory capacity.Fibrosis: recent advances in myofibroblast biology and new therapeutic perspectives.Masters and servants of the force: the role of matrix adhesions in myofibroblast force perception and transmission.The mechanical environment modulates intracellular calcium oscillation activities of myofibroblasts.A key role for NOX4 in epithelial cell death during development of lung fibrosis.Mechanisms of force generation and transmission by myofibroblasts.Temporal and Molecular Analyses of Cardiac Extracellular Matrix Remodeling following Pressure Overload in Adiponectin Deficient MiceHyperglycemia Interacts with Ischemia in a Synergistic Way on Wound Repair and Myofibroblast Differentiation.The NH2-terminal peptide of alpha-smooth muscle actin inhibits force generation by the myofibroblast in vitro and in vivo.Marching at the front and dragging behind: differential alphaVbeta3-integrin turnover regulates focal adhesion behaviorCulture of primary bovine chondrocytes on a continuously expanding surface inhibits dedifferentiationHic-5 is required for myofibroblast differentiation by regulating mechanically dependent MRTF-A nuclear accumulationThe 22nd annual meeting of the European Tissue Repair Society (ETRS) in Athens, Greece.Formation and function of the myofibroblast during tissue repair.Strategies to overcome the hurdles to treat fibrosis, a major unmet clinical need.Preclinical Models of Wound Healing: Is Man the Model? Proceedings of the Wound Healing Society Symposium.Myofibroblasts work best under stress.Myofibroblast development is characterized by specific cell-cell adherens junctions.The myofibroblast: paradigm for a mechanically active cell.The myofibroblast matrix: implications for tissue repair and fibrosis.Regulation of myofibroblast activities: calcium pulls some strings behind the scene.Mechanical induction of gene expression in connective tissue cells.Matrix mechanics and regulation of the fibroblast phenotype.Fibrogenic fibroblasts increase intercellular adhesion strength by reinforcing individual OB-cadherin bonds.
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description
hulumtues
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researcher
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wetenschapper
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հետազոտող
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name
Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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type
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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Boris Hinz
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P1153
7005792281
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P31
P496
0000-0002-0526-393X