Diesel exhaust enhances influenza virus infections in respiratory epithelial cells. - Wikidata - wikimedia - TriplyDB

Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

http://www.wikidata.org/entity/Q40445815

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Fine particulate matter in acute exacerbation of COPD N-acetylcysteine synergizes with oseltamivir in protecting mice from lethal influenza infection Role of oxidative stress on diesel-enhanced influenza infection in mice Modification by influenza on health effects of air pollution in Hong Kong Exceptionally high mortality rate of the 1918 influenza pandemic in the Brazilian naval fleet.Exposure to ozone modulates human airway protease/antiprotease balance contributing to increased influenza A infection Disruption of microRNA expression in human airway cells by diesel exhaust particles is linked to tumorigenesis-associated pathways Respiratory syncytial virus infection reduces lung inflammation and fibrosis in mice exposed to vanadium pentoxide.Reduced expression of IRF7 in nasal epithelial cells from smokers after infection with influenza.Air pollution and airway disease.Localization of type I interferon receptor limits interferon-induced TLR3 in epithelial cells.Exposure to combustion generated environmentally persistent free radicals enhances severity of influenza virus infection Methamphetamine reduces human influenza A virus replication.Tobacco smoke exposure and altered nasal responses to live attenuated influenza virus.Nrf2 expression modifies influenza A entry and replication in nasal epithelial cells.Epithelial cells, the "switchboard" of respiratory immune defense responses: effects of air pollutants.Epithelial cells from smokers modify dendritic cell responses in the context of influenza infection.Alteration of the nasal responses to influenza virus by tobacco smoke Selenium deficiency alters epithelial cell morphology and responses to influenza Influenza enhances caspase-1 in bronchial epithelial cells from asthmatic volunteers and is associated with pathogenesis.The Environmental Pollutant Cadmium Promotes Influenza Virus Replication in MDCK Cells by Altering Their Redox State Diesel exhaust particles modify natural killer cell function and cytokine release.Diesel exposure suppresses natural killer cell function and resolution of eosinophil inflammation: a randomized controlled trial of exposure in allergic rhinitics.Toxicological evaluation of realistic emission source aerosols (TERESA): introduction and overview.Air pollution and respiratory viral infection.Implication of scavenger receptors in the interactions between diesel exhaust particles and immature or mature dendritic cells.Exacerbation of allergic inflammation in mice exposed to diesel exhaust particles prior to viral infection.Epithelial NF-κB orchestrates house dust mite-induced airway inflammation, hyperresponsiveness, and fibrotic remodeling Culturing of human nasal epithelial cells at the air liquid interface.Live attenuated influenza vaccine strains elicit a greater innate immune response than antigenically-matched seasonal influenza viruses during infection of human nasal epithelial cell cultures.The role of oxidative stress and innate immunity in O(3) and endotoxin-induced human allergic airway disease.Response to infections in patients with asthma and atopic disease: an epiphenomenon or reflection of host susceptibility?Epithelial function and dysfunction in asthma.Ozone-derived Oxysterols Affect Liver X Receptor (LXR) Signaling: A POTENTIAL ROLE FOR LIPID-PROTEIN ADDUCTS.Urban particulate matter increases human airway epithelial cell IL-1β secretion following scratch wounding and H1N1 influenza A exposure in vitro.Endoplasmic reticulum stress mediates house dust mite-induced airway epithelial apoptosis and fibrosis.Haze is a risk factor contributing to the rapid spread of respiratory syncytial virus in children.Exposure to silver nanoparticles affects viability and function of natural killer cells, mostly via the release of ions.Diesel exhaust enhances virus- and poly(I:C)-induced Toll-like receptor 3 expression and signaling in respiratory epithelial cells.Novel applications for a noninvasive sampling method of the nasal mucosa.

P2860

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P2860

Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

http://www.wikidata.org/entity/Q40445815

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

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Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

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Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

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Toxicological Sciences

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Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

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Ilona Jaspers

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Johnny L Carson

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Jonathan M Ciencewicki

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Luisa E Brighton

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Melinda A Beck

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Michael C Madden

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Wenli Zhang

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P2860

Influenza virus-induced alterations of cytochrome P-450 enzyme activities following exposure of mice to coal and diesel particulates.

Influenza virus infection in mice after exposure to coal dust and diesel engine emissions

Diesel exhaust particles suppress macrophage function and slow the pulmonary clearance of listeria monocytogenes in rats

Effect of exposure to diesel exhaust particles on the suspectibility of the lung to infection

Suppression of cell-mediated immune responses to Listeria infection by repeated exposure to diesel exhaust particles

Development of a real-time reverse transcriptase PCR assay for type A influenza virus and the avian H5 and H7 hemagglutinin subtypes

Antiviral actions of interferons

Diesel exhaust and asthma: hypotheses and molecular mechanisms of action.

Localization of lung surfactant protein D on mucosal surfaces in human tissues

Inhibition of influenza infection by glutathione

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990-1002

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10.1093/TOXSCI/KFI141

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2

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85

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