Reactive oxygen species from smooth muscle mitochondria initiate cold-induced constriction of cutaneous arteries.
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Targeting mitochondrial reactive oxygen species as novel therapy for inflammatory diseases and cancersCyclic AMP acts through Rap1 and JNK signaling to increase expression of cutaneous smooth muscle alpha2C-adrenoceptorsEstrogen increases smooth muscle expression of alpha2C-adrenoceptors and cold-induced constriction of cutaneous arteriesAcute vibration increases alpha(2C)-adrenegic smooth muscle constriction and alters thermosensitivity of cutaneous arteriesRegulation of vascular reactivity in scleroderma: new insights into Raynaud's phenomenonDetection of reactive oxygen species in isolated, perfused lungs by electron spin resonance spectroscopy.Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosisRaynaud's Phenomenon: A Brief Review of the Underlying MechanismsLocal thermal control of the human cutaneous circulationTime-dependent and somatically acquired mitochondrial DNA mutagenesis and respiratory chain dysfunction in a scleroderma model of lung fibrosisCyclic AMP-Rap1A signaling activates RhoA to induce α(2c)-adrenoceptor translocation to the cell surface of microvascular smooth muscle cellsIntracellular α(2C)-adrenoceptors: storage depot, stunted development or signaling domain?Cyclic AMP-Rap1A signaling mediates cell surface translocation of microvascular smooth muscle α2C-adrenoceptors through the actin-binding protein filamin-2In silico modeling of human α2C-adrenoreceptor interaction with filamin-2Aging and the control of human skin blood flowHuman skeletal muscle feed arteries studied in vitro: the effect of temperature on α(1)-adrenergic responsivenessLocal regulation of skin blood flow during cooling involving presynaptic P2 purinoceptors in rats.Angiotensin II type 1 receptor blockade corrects cutaneous nitric oxide deficit in postural tachycardia syndromeEffect of elevated local temperature on cutaneous vasoconstrictor responsiveness in humansThe involvement of nitric oxide in the cutaneous vasoconstrictor response to local cooling in humans.Cold-induced cutaneous vasoconstriction is mediated by Rho kinase in vivo in human skin.Rho kinase-mediated local cold-induced cutaneous vasoconstriction is augmented in aged human skin.Role of sensory nerves in the cutaneous vasoconstrictor response to local cooling in humans.Role of alpha2C-adrenoceptors in the reduction of skin blood flow induced by local cooling in mice.The role of baseline in the cutaneous vasoconstrictor responses during combined local and whole body cooling in humans.Reflex vasoconstriction in aged human skin increasingly relies on Rho kinase-dependent mechanisms during whole body coolingLocal ascorbate administration inhibits the adrenergic vasoconstrictor response to local cooling in the human skin.Fasudil mesylate protects PC12 cells from oxidative stress injury via the Bax-mediated pathway.Noninvasive cutaneous blood flow as a response predictor for visible light therapy on segmental vitiligo: a prospective pilot study.Recent achievements in the management of Raynaud's phenomenon.Upregulation of inducible nitric oxide synthase contributes to attenuated cutaneous vasodilation in essential hypertensive humans.Microcirculation response to local cooling in patients with Huntington's disease.Increased leukocyte rho kinase (ROCK) activity and endothelial dysfunction in cigarette smokersReactive oxygen species are involved in regulating alpha1-adrenoceptor-activated vascular smooth muscle contraction.Oral vitamin C enhances the adrenergic vasoconstrictor response to local cooling in human skin.Thermal provocation to evaluate microvascular reactivity in human skin.Mitochondria-derived reactive oxygen species dilate cerebral arteries by activating Ca2+ sparks.Melanopsin mediates light-dependent relaxation in blood vessels.Whole-body cryostimulation--potential beneficial treatment for improving antioxidant capacity in healthy men--significance of the number of sessions.Cold sensitivity of TRPA1 is unveiled by the prolyl hydroxylation blockade-induced sensitization to ROS
P2860
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P2860
Reactive oxygen species from smooth muscle mitochondria initiate cold-induced constriction of cutaneous arteries.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Reactive oxygen species from s ...... riction of cutaneous arteries.
@en
type
label
Reactive oxygen species from s ...... riction of cutaneous arteries.
@en
prefLabel
Reactive oxygen species from s ...... riction of cutaneous arteries.
@en
P2093
P2860
P1476
Reactive oxygen species from s ...... riction of cutaneous arteries.
@en
P2093
P2860
P304
P356
10.1152/AJPHEART.01305.2004
P577
2005-03-11T00:00:00Z