Vitamin C protects HL60 and U266 cells from arsenic toxicity. - Wikidata - wikimedia - TriplyDB

Vitamin C protects HL60 and U266 cells from arsenic toxicity.

Vitamin C protects HL60 and U266 cells from arsenic toxicity.

http://www.wikidata.org/entity/Q40464464

about

Arsenic trioxide - An old drug rediscovered.Arsenic trioxide and ascorbic acid demonstrate promising activity against primary human CLL cells in vitro.Protective efficacy of vitamins C and E on p,p'-DDT-induced cytotoxicity via the ROS-mediated mitochondrial pathway and NF-κB/FasL pathway.Vitamin C antagonizes the cytotoxic effects of antineoplastic drugs.Vitamin C suppresses cell death in MCF-7 human breast cancer cells induced by tamoxifen.Arsenic trioxide-based therapy in relapsed/refractory multiple myeloma patients: a meta-analysis and systematic review.As(2)O (3) oxidation by vitamin C: cell culture studies.Effects of nutritional measures on toenail arsenic concentration as a biomarker of arsenic exposure.Trolox enhances the anti-lymphoma effects of arsenic trioxide, while protecting against liver toxicity.Efficacy and safety of melphalan, arsenic trioxide and ascorbic acid combination therapy in patients with relapsed or refractory multiple myeloma: a prospective, multicentre, phase II, single-arm study.Arsenical keratosis caused by medication: a case report and literature.Green tea and vitamin C ameliorate some neuro-functional and biochemical signs of arsenic toxicity in rats.

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Vitamin C protects HL60 and U266 cells from arsenic toxicity.

http://www.wikidata.org/entity/Q40464464

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2005 nî lūn-bûn

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Vitamin C protects HL60 and U266 cells from arsenic toxicity.

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Vitamin C protects HL60 and U266 cells from arsenic toxicity.

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Vitamin C protects HL60 and U266 cells from arsenic toxicity.

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Vitamin C protects HL60 and U266 cells from arsenic toxicity.

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David W Golde

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George Stratis

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Juan M Cárcamo

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Nicos Karasavvas

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P2860

A family of mammalian Na+-dependent L-ascorbic acid transporters

Does vitamin C act as a pro-oxidant under physiological conditions?

United States multicenter study of arsenic trioxide in relapsed acute promyelocytic leukemia

The PML-RAR alpha fusion mRNA generated by the t(15;17) translocation in acute promyelocytic leukemia encodes a functionally altered RAR

Arsenic trioxide sensitivity is associated with low level of glutathione in cancer cells

Cytotoxicity of ascorbate, lipoic acid, and other antioxidants in hollow fibre in vitro tumours

Arsenic trioxide induces apoptosis in neuroblastoma cell lines through the activation of caspase 3 in vitro

Arsenic trioxide-induced apoptosis and differentiation are associated respectively with mitochondrial transmembrane potential collapse and retinoic acid signaling pathways in acute promyelocytic leukemia

Arsenic trioxide selectively induces acute promyelocytic leukemia cell apoptosis via a hydrogen peroxide-dependent pathway

Vitamin C crosses the blood-brain barrier in the oxidized form through the glucose transporters

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4004-4012

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10.1182/BLOOD-2003-03-0772

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