Cell-protection mechanism through autophagy in HGFs/S. mitis co-culture treated with Chitlac-nAg.

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Cell-protection mechanism through autophagy in HGFs/S. mitis co-culture treated with Chitlac-nAg.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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name

Cell-protection mechanism thro ...... ture treated with Chitlac-nAg.

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label

Cell-protection mechanism thro ...... ture treated with Chitlac-nAg.

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Cell-protection mechanism thro ...... ture treated with Chitlac-nAg.

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P1476

Cell-protection mechanism thro ...... lture treated with Chitlac-nAg

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P2093

Amelia Cataldi

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Andrea Travan

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Domenico Bosco

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Monica Rapino

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Roberta Di Pietro

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Sergio Paoletti

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Silvia Sancilio

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Valentina Di Valerio

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Viviana di Giacomo

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P2860

Phosphatidylserine in addition to phosphatidylethanolamine is an in vitro target of the mammalian Atg8 modifiers, LC3, GABARAP, and GATE-16

It's a cell-eat-cell world: autophagy and phagocytosis.

Methods in mammalian autophagy research

Regulated portals of entry into the cell

Silver-polysaccharide nanocomposite antimicrobial coatings for methacrylic thermosets.

Autophagy: cellular and molecular mechanisms.

Autophagy regulates TGF-β expression and suppresses kidney fibrosis induced by unilateral ureteral obstruction.

Biological responses of human gingival fibroblasts (HGFs) in an innovative co-culture model with Streptococcus mitis to thermosets coated with a silver polysaccharide antimicrobial system

Exposure to silver nanoparticles induces size- and dose-dependent oxidative stress and cytotoxicity in human colon carcinoma cells.

Silver-polysaccharide antimicrobial nanocomposite coating for methacrylic surfaces reduces Streptococcus mutans biofilm formation in vitro.

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s10856-016-5803-5

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186

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scholarly article

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10.1007/S10856-016-5803-5

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P478

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Mara Di Giulio

M Gallorini

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2016-10-27T00:00:00Z

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1007661194

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P698

27787811

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P921

autophagy