The rejoining of double-strand breaks in DNA by human cell extracts
about
The human DNA ligase III gene encodes nuclear and mitochondrial proteinsThe role of DNA polymerase activity in human non-homologous end joiningThe SET domain protein Metnase mediates foreign DNA integration and links integration to nonhomologous end-joining repairDeficient DNA end joining activity in extracts from fanconi anemia fibroblasts.Nonhomologous DNA end joining in cell-free systems.In vitro repair of complex unligatable oxidatively induced DNA double-strand breaks by human cell extracts.Multiple pathways for repair of DNA double-strand breaks in mammalian chromosomesA double-strand break in a chromosomal LINE element can be repaired by gene conversion with various endogenous LINE elements in mouse cellsIn vitro non-homologous DNA end joining assays--the 20th anniversary.Anti-apoptotic protein BCL2 down-regulates DNA end joining in cancer cells.Directional recombination is initiated at a double strand break in human nuclear extracts.Fidelity of end joining in mammalian episomes and the impact of Metnase on joint processing.RecQ helicases: suppressors of tumorigenesis and premature aging.Base damage immediately upstream from double-strand break ends is a more severe impediment to nonhomologous end joining than blocked 3'-terminiChromosomal double-strand breaks induce gene conversion at high frequency in mammalian cellsMechanisms of overlap formation in nonhomologous DNA end joining.Activation of a system for the joining of nonhomologous DNA ends during Xenopus egg maturation.Repair of sequence-specific 125I-induced double-strand breaks by nonhomologous DNA end joining in mammalian cell-free extracts.Effects of mutations of RAD50, RAD51, RAD52, and related genes on illegitimate recombination in Saccharomyces cerevisiae.Development of a novel rapid assay to assess the fidelity of DNA double-strand-break repair in human tumour cells.Hdf1, a yeast Ku-protein homologue, is involved in illegitimate recombination, but not in homologous recombination.The joining of non-complementary DNA double-strand breaks by mammalian extractsNon-homologous DNA end joining in plant cells is associated with deletions and filler DNA insertions.A modified single-strand annealing model best explains the joining of DNA double-strand breaks mammalian cells and cell extracts.Nonhomologous DNA end joining of synthetic hairpin substrates in Xenopus laevis egg extracts.Nonhomologous DNA end joining in Schizosaccharomyces pombe efficiently eliminates DNA double-strand-breaks from haploid sequences.Repair of a specific double-strand break generated within a mammalian chromosome by yeast endonuclease I-SceI.Multiple components are involved in the efficient joining of double stranded DNA breaks in human cell extracts.A mechanism for deletion formation in DNA by human cell extracts: the involvement of short sequence repeats.Cellular radiosensitivity in ataxia-telangiectasia.Molecular determinants of radiosensitivity in mammalian cells.The study of responses to 'model' DNA breaks induced by restriction endonucleases in cells and cell-free systems: achievements and difficulties.Weiss Lecture. Effects of radiations of different qualities on cells: molecular mechanisms of damage and repair.Increased error-prone non homologous DNA end-joining--a proposed mechanism of chromosomal instability in Bloom's syndrome.Expression of ATM in ataxia telangiectasia fibroblasts rescues defects in DNA double-strand break repair in nuclear extracts.Enhancement of frequencies of restriction endonuclease-induced chromatid breaks by arabinoside adenine in normal human and ataxia telangiectasia cells.Hypersensitivity of ataxia telangiectasia fibroblasts to ionizing radiation is associated with a repair deficiency of DNA double-strand breaks.Radiosensitivity and double-strand break rejoining in tumorigenic and non-tumorigenic human epithelial cell lines.Enhanced chromosomal response of ataxia-telangiectasia cells to specific types of DNA double-strand breaks.Characterization of DNA end joining in a mammalian cell nuclear extract: junction formation is accompanied by nucleotide loss, which is limited and uniform but not site specific.
P2860
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P2860
The rejoining of double-strand breaks in DNA by human cell extracts
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
The rejoining of double-strand breaks in DNA by human cell extracts
@en
type
label
The rejoining of double-strand breaks in DNA by human cell extracts
@en
prefLabel
The rejoining of double-strand breaks in DNA by human cell extracts
@en
P2093
P2860
P356
P1476
The rejoining of double-strand breaks in DNA by human cell extracts
@en
P2093
P2860
P304
P356
10.1093/NAR/18.21.6205
P577
1990-11-01T00:00:00Z