Lipoxin A4, a 5-lipoxygenase pathway metabolite, modulates immune response during acute respiratory tularemia.
about
Necroptotic debris including damaged mitochondria elicits sepsis-like syndrome during late-phase tularemia.Host-based lipid inflammation drives pathogenesis in Francisella infection.Arachidonic acid and other unsaturated fatty acids and some of their metabolites function as endogenous antimicrobial molecules: A review
P2860
Lipoxin A4, a 5-lipoxygenase pathway metabolite, modulates immune response during acute respiratory tularemia.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Lipoxin A4, a 5-lipoxygenase p ...... g acute respiratory tularemia.
@en
type
label
Lipoxin A4, a 5-lipoxygenase p ...... g acute respiratory tularemia.
@en
prefLabel
Lipoxin A4, a 5-lipoxygenase p ...... g acute respiratory tularemia.
@en
P2093
P2860
P356
P1476
Lipoxin A4, a 5-lipoxygenase p ...... g acute respiratory tularemia.
@en
P2093
Alireza Arabshahi
Anju Singh
Florin Marcel Musteata
Jeevan Prasain
Rose Bartiss
Stephen Barnes
Tabassum Rahman
Timothy J Sellati
P2860
P304
P356
10.1189/JLB.4A0815-365RR
P577
2016-09-14T00:00:00Z