Circulating Calreticulin Is Increased in Myelofibrosis: Correlation with Interleukin-6 Plasma Levels, Bone Marrow Fibrosis, and Splenomegaly

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Calreticulin Fragment 39-272 Promotes B16 Melanoma Malignancy through Myeloid-Derived Suppressor Cells In Vivo

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Circulating Calreticulin Is Increased in Myelofibrosis: Correlation with Interleukin-6 Plasma Levels, Bone Marrow Fibrosis, and Splenomegaly

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2016 nî lūn-bûn

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Circulating Calreticulin Is In ...... row Fibrosis, and Splenomegaly

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Circulating Calreticulin Is In ...... row Fibrosis, and Splenomegaly

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Circulating Calreticulin Is In ...... row Fibrosis, and Splenomegaly

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Daria Sollazzo

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Michele Cavo

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Nicola Vianelli

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Simona Luatti

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The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes

Somatic CALR mutations in myeloproliferative neoplasms with nonmutated JAK2

MPNs as Inflammatory Diseases: The Evidence, Consequences, and Perspectives

Inflammation as a Driver of Clonal Evolution in Myeloproliferative Neoplasm

Impact of Inflammation on Myeloproliferative Neoplasm Symptom Development

Pathogenesis of Myeloproliferative Neoplasms: Role and Mechanisms of Chronic Inflammation

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Circulating interleukin (IL)-8, IL-2R, IL-12, and IL-15 levels are independently prognostic in primary myelofibrosis: a comprehensive cytokine profiling study.

Integrated genomic analysis illustrates the central role of JAK-STAT pathway activation in myeloproliferative neoplasm pathogenesis.

Somatic mutations of calreticulin in myeloproliferative neoplasms.

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Circulating Calreticulin Is Increased in Myelofibrosis: Correlation with Interleukin-6 Plasma Levels, Bone Marrow Fibrosis, and Splenomegaly

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P2860

P2860

Circulating Calreticulin Is Increased in Myelofibrosis: Correlation with Interleukin-6 Plasma Levels, Bone Marrow Fibrosis, and Splenomegaly

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