Null mutation of gp91phox reduces muscle membrane lysis during muscle inflammation in mice.
about
Modulating exercise-induced hormesis: Does less equal more?Insights into the molecular etiology of exercise-induced inflammation: opportunities for optimizing performanceSkeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse.Regulatory interactions between muscle and the immune system during muscle regenerationInterleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype.Macrophages promote muscle membrane repair and muscle fibre growth and regeneration during modified muscle loading in mice in vivo.IL-10 triggers changes in macrophage phenotype that promote muscle growth and regeneration.The role of neutrophils in injury and repair following muscle stretch.The emerging biology of muscle stem cells: implications for cell-based therapies.Dystrophin-deficient cardiomyopathy in mouse: expression of Nox4 and Lox are associated with fibrosis and altered functional parameters in the heartExacerbation of pathology by oxidative stress in respiratory and locomotor muscles with Duchenne muscular dystrophy.Contribution of oxidative stress to pathology in diaphragm and limb muscles with Duchenne muscular dystrophy.Mechanisms of muscle injury, repair, and regeneration.Urinary biomarkers of physical activity: candidates and clinical utility.Oxidative stress in muscular dystrophy: from generic evidence to specific sources and targets.Treatment with selectin blocking antibodies after lengthening contractions of mouse muscle blunts neutrophil accumulation but does not reduce damage.Muscle damage and inflammation during recovery from exercise.The effect of polyphenols on cytokine and granulocyte response to resistance exercise.From innate to adaptive immune response in muscular dystrophies and skeletal muscle regeneration: the role of lymphocytesThe Effect of Reloading on Disuse Muscle Atrophy: Time Course of Hypertrophy and Regeneration Focusing on the Myofiber Cross-sectional Area and Myonuclear Change.Effect of anti-inflammatory and antioxidant drugs on the long-term repair of severely injured mouse skeletal muscle.Null mutation of myeloperoxidase in mice prevents mechanical activation of neutrophil lysis of muscle cell membranes in vitro and in vivo.N-Acetylcysteine ameliorates skeletal muscle pathophysiology in mdx mice.[Buruli ulcer - beyond the skin, impact on skeletal muscle].Changes in inflammatory and oxidative stress factors and the protein synthesis pathway in injured skeletal muscle after contusion.Neutrophil and monocyte responses to downhill running: Intracellular contents of MPO, IL-6, IL-10, pstat3, and SOCS3.
P2860
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P2860
Null mutation of gp91phox reduces muscle membrane lysis during muscle inflammation in mice.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Null mutation of gp91phox redu ...... g muscle inflammation in mice.
@en
type
label
Null mutation of gp91phox redu ...... g muscle inflammation in mice.
@en
prefLabel
Null mutation of gp91phox redu ...... g muscle inflammation in mice.
@en
P2860
P1476
Null mutation of gp91phox redu ...... g muscle inflammation in mice.
@en
P2093
Hal X Nguyen
James G Tidball
P2860
P304
P356
10.1113/JPHYSIOL.2003.051912
P407
P577
2003-10-10T00:00:00Z