Histone acetylation regulates epithelial IL-8 release mediated by oxidative stress from environmental particles.
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Principles for characterizing the potential human health effects from exposure to nanomaterials: elements of a screening strategyConcordance between in vitro and in vivo dosimetry in the proinflammatory effects of low-toxicity, low-solubility particles: the key role of the proximal alveolar regionThe limits of testing particle-mediated oxidative stress in vitro in predicting diverse pathologies; relevance for testing of nanoparticlesPossible molecular mechanisms linking air pollution and asthma in childrenGenetic and epigenetic influence on the response to environmental particulate matterThe genetic and epigenetic landscapes of the epithelium in asthmaThe pro-inflammatory effects of low-toxicity low-solubility particles, nanoparticles and fine particles, on epithelial cells in vitro: the role of surface areaEnvironmental epigenetics of asthma: an updateEpigenetic Regulation in Particulate Matter-Mediated Cardiopulmonary Toxicities: A Systems Biology PerspectiveEnvironmental chemical exposures and human epigeneticsHypertensive rats are susceptible to TLR4-mediated signaling following exposure to combustion source particulate matterEpigenetics underpinning the regulation of the CXC (ELR+) chemokines in non-small cell lung cancerNanoparticles-a thoracic toxicology perspectiveEnvironmental exposures and gene regulation in disease etiologyStudy of oxidative stress biomarkers in chronic obstructive pulmonary disease and their correlation with disease severity in north Indian population cohortNF-κB inducing kinase, NIK mediates cigarette smoke/TNFα-induced histone acetylation and inflammation through differential activation of IKKs.Transcriptional regulation of cyclooxygenase-2 in response to proteasome inhibitors involves reactive oxygen species-mediated signaling pathway and recruitment of CCAAT/enhancer-binding protein delta and CREB-binding proteinAir pollution and cancer: biomarker studies in human populations.Potential biological applications of bio-based anacardic acids and their derivatives.Risk of nonlower respiratory serious adverse events following COPD exacerbations in the 4-year UPLIFT® trial.Brd4 is essential for IL-1β-induced inflammation in human airway epithelial cellsEnvironmental and epigenetic effects upon preimplantation embryo metabolism and development.Inhalable metal-rich air particles and histone H3K4 dimethylation and H3K9 acetylation in a cross-sectional study of steel workers.Cytotoxic effect of curcumin on malaria parasite Plasmodium falciparum: inhibition of histone acetylation and generation of reactive oxygen species.Recent advances in underlying pathologies provide insight into interleukin-8 expression-mediated inflammation and angiogenesis.Mitogen- and stress-activated kinase 1 (MSK1) regulates cigarette smoke-induced histone modifications on NF-κB-dependent genesInhaled particles and lung cancer, part B: paradigms and risk assessment.Stress-induced cell reprogramming. A role for global genome regulation?Elevated H3K18 acetylation in airway epithelial cells of asthmatic subjects.Characterization of genome-wide H3K27ac profiles reveals a distinct PM2.5-associated histone modification signature.Developmental origins of adult disease.The role of oxidative stress in the pathogenesis of COPD: implications for therapy.Temporal relationship between air pollutants and hospital admissions for chronic obstructive pulmonary disease in Hong Kong.Serum concentrations of HGF and IL-8 in patients with active Graves' orbitopathy before and after methylprednisolone therapyCombined inhaled diesel exhaust particles and allergen exposure alter methylation of T helper genes and IgE production in vivo.Developmental origins of beta-cell failure in type 2 diabetes: the role of epigenetic mechanisms.Role of metabolic programming in the pathogenesis of beta-cell failure in postnatal life.Biological targets for therapeutic interventions in COPD: clinical potentialThe histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activation.Loss of CFTR results in reduction of histone deacetylase 2 in airway epithelial cells.
P2860
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P2860
Histone acetylation regulates epithelial IL-8 release mediated by oxidative stress from environmental particles.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Histone acetylation regulates ...... from environmental particles.
@en
type
label
Histone acetylation regulates ...... from environmental particles.
@en
prefLabel
Histone acetylation regulates ...... from environmental particles.
@en
P2093
P2860
P1476
Histone acetylation regulates ...... from environmental particles.
@en
P2093
Irfan Rahman
Kenneth Donaldson
Peter S Gilmour
P2860
P304
P356
10.1152/AJPLUNG.00277.2002
P577
2002-11-22T00:00:00Z