Inorganic arsenite-induced malignant transformation of human prostate epithelial cells. - Wikidata - wikimedia - TriplyDB

Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

http://www.wikidata.org/entity/Q40682771

about

Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cells Association of chromosomal alterations with arsenite-induced tumorigenicity of human HaCaT keratinocytes in nude mice.Acquisition of androgen independence by human prostate epithelial cells during arsenic-induced malignant transformation.Distinct gene expression profiles in immortalized human urothelial cells exposed to inorganic arsenite and its methylated trivalent metabolites Arsenic in drinking water and prostate cancer in Illinois counties: an ecologic study Epigenetic changes during cell transformation The microRNA-200 family: small molecules with novel roles in cancer development, progression and therapy Inorganic arsenic and human prostate cancer Neovascularization and angiogenic gene expression following chronic arsenic exposure in mice Environmental arsenic exposure and serum matrix metalloproteinase-9 Arsenic-induced malignant transformation of human keratinocytes: involvement of Nrf2 Chronic inorganic arsenic exposure in vitro induces a cancer cell phenotype in human peripheral lung epithelial cells Akt activation is responsible for enhanced migratory and invasive behavior of arsenic-transformed human bronchial epithelial cells The Use of Chemical-Chemical Interaction and Chemical Structure to Identify New Candidate Chemicals Related to Lung Cancer Methylarsonous acid causes oxidative DNA damage in cells independent of the ability to biomethylate inorganic arsenic Enhanced glutathione biosynthetic capacity promotes resistance to As3+-induced apoptosis.Arsenic exposure transforms human epithelial stem/progenitor cells into a cancer stem-like phenotype.TPL2/COT/MAP3K8 (TPL2) activation promotes androgen depletion-independent (ADI) prostate cancer growth.Arsenic-specific stem cell selection during malignant transformation.Monomethylarsonous acid produces irreversible events resulting in malignant transformation of a human bladder cell line following 12 weeks of low-level exposure Influence of arsenate and arsenite on signal transduction pathways: an update MicroRNA-200b suppresses arsenic-transformed cell migration by targeting protein kinase Cα and Wnt5b-protein kinase Cα positive feedback loop and inhibiting Rac1 activation Methylated arsenicals: the implications of metabolism and carcinogenicity studies in rodents to human risk assessment.Genetic deletion of LXRα prevents arsenic-enhanced atherosclerosis, but not arsenic-altered plaque composition.Silencing KRAS overexpression in arsenic-transformed prostate epithelial and stem cells partially mitigates malignant phenotype.Activation and up-regulation of translation initiation factor 4B contribute to arsenic-induced transformation Mechanisms of acquired androgen independence during arsenic-induced malignant transformation of human prostate epithelial cells.Differential DNA methylation profile of key genes in malignant prostate epithelial cells transformed by inorganic arsenic or cadmium Arsenic-transformed malignant prostate epithelia can convert noncontiguous normal stem cells into an oncogenic phenotype.Neoplastic transformation of human small airway epithelial cells induced by arsenic.Chronic cadmium exposure in vitro causes acquisition of multiple tumor cell characteristics in human pancreatic epithelial cells.Mitochondrial alteration in malignantly transformed human small airway epithelial cells induced by α-particles Elevated concentrations of serum matrix metalloproteinase-2 and -9 and their associations with circulating markers of cardiovascular diseases in chronic arsenic-exposed individuals.Agglomerates of aberrant DNA methylation are associated with toxicant-induced malignant transformation.Lung inflammation biomarkers and lung function in children chronically exposed to arsenic Arsenite activates NFκB through induction of C-reactive protein.Interplay between cellular methyl metabolism and adaptive efflux during oncogenic transformation from chronic arsenic exposure in human cells Arsenic upregulates MMP-9 and inhibits wound repair in human airway epithelial cells Epithelial to mesenchymal transition in arsenic-transformed cells promotes angiogenesis through activating β-catenin-vascular endothelial growth factor pathway.Recruitment of normal stem cells to an oncogenic phenotype by noncontiguous carcinogen-transformed epithelia depends on the transforming carcinogen.

P2860

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P2860

Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

http://www.wikidata.org/entity/Q40682771

description

2002 nî lūn-bûn

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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2002年论文

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2002年论文

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name

Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

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type

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Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

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prefLabel

Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

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Journal of the National Cancer Institute

P1476

Inorganic arsenite-induced malignant transformation of human prostate epithelial cells.

@en

P2093

Bhalchandra A Diwan

http://www.w3.org/2001/XMLSchema#string

Eduardo M Brambila

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Michael P Waalkes

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Mukta M Webber

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William E Achanzar

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P2860

Cancer statistics, 2002

Association of arsenic-induced malignant transformation with DNA hypomethylation and aberrant gene expression

Genetic events associated with arsenic-induced malignant transformation: applications of cDNA microarray technology.

Recent advances in arsenic carcinogenesis: modes of action, animal model systems, and methylated arsenic metabolites.

Arsenite is a cocarcinogen with solar ultraviolet radiation for mouse skin: an animal model for arsenic carcinogenesis.

Genetic toxicology of a paradoxical human carcinogen, arsenic: a review.

Drinking water arsenic in Utah: A cohort mortality study.

Association of c-myc overexpression and hyperproliferation with arsenite-induced malignant transformation.

Hormones and cancer in humans.

Acinar differentiation by non-malignant immortalized human prostatic epithelial cells and its loss by malignant cells.

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1888-1891

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scholarly article

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10.1093/JNCI/94.24.1888

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24

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94

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2002-12-01T00:00:00Z

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10984469

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12488483

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