Intracellular localization of keratinocyte Fas ligand explains lack of cytolytic activity under physiological conditions. - Wikidata - wikimedia - TriplyDB

Intracellular localization of keratinocyte Fas ligand explains lack of cytolytic activity under physiological conditions.

Intracellular localization of keratinocyte Fas ligand explains lack of cytolytic activity under physiological conditions.

http://www.wikidata.org/entity/Q40684481

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Toxic epidermal necrolysis and Stevens-Johnson syndrome Toxic epidermal necrolysis Cytotoxic proteins and therapeutic targets in severe cutaneous adverse reactions Amniotic membrane transplantation as a new therapy for the acute ocular manifestations of Stevens-Johnson syndrome and toxic epidermal necrolysis.Role of Fas/FasL in regulation of inflammation in vaginal tissue during HSV-2 infection.Killer B lymphocytes and their fas ligand positive exosomes as inducers of immune tolerance Regulating TRAIL receptor-induced cell death at the membrane : a deadly discussion Phenytoin-induced Lyell's syndrome.Keratinocyte apoptosis in epidermal development and disease.APRIL secreted by neutrophils binds to heparan sulfate proteoglycans to create plasma cell niches in human mucosa.Photosensitivity, apoptosis, and cytokines in the pathogenesis of lupus erythematosus: a critical review.Immunological response in Stevens-Johnson syndrome and toxic epidermal necrolysis.Adverse cutaneous drug eruptions: current understanding.Immunologic Mediators in Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis.RUNX3 cooperates with FoxO3a to induce apoptosis in gastric cancer cells.The expression of selected proapoptotic molecules in dermatitis herpetiformis.Possible role of Fas/Fas ligand-mediated apoptosis in the pathogenesis of fixed drug eruption.Soluble Fas Ligand Is Essential for Blister Formation in Pemphigus.

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P2860

Intracellular localization of keratinocyte Fas ligand explains lack of cytolytic activity under physiological conditions.

http://www.wikidata.org/entity/Q40684481

description

2002 nî lūn-bûn

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2002年の論文

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2002年論文

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2002年论文

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name

Intracellular localization of ...... nder physiological conditions.

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Intracellular localization of ...... nder physiological conditions.

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Intracellular localization of ...... nder physiological conditions.

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P1433

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P1433

Journal of Biological Chemistry

P1476

Intracellular localization of ...... under physiological conditions

@en

P2093

Alain Limat

http://www.w3.org/2001/XMLSchema#string

Isabelle Viard-Leveugle

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Jean-Hilaire Saurat

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Jürg Tschopp

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Paolo Meda

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Roberto R Bullani

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P2860

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

Death receptors in cutaneous biology and disease.

Inhibition of toxic epidermal necrolysis by blockade of CD95 with human intravenous immunoglobulin.

Fas and Fas ligand in embryos and adult mice: ligand expression in several immune-privileged tissues and coexpression in adult tissues characterized by apoptotic cell turnover.

Metalloproteinase-mediated release of human Fas ligand

P304

16183-16188

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scholarly article

P356

10.1074/JBC.M212188200

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18

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P478

278

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Olivier Micheau

P577

2002-12-07T00:00:00Z

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P698

12473659

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