Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis.
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Myocardial MiR-30 downregulation triggered by doxorubicin drives alterations in β-adrenergic signaling and enhances apoptosisDoxorubicin activates nuclear factor of activated T-lymphocytes and Fas ligand transcription: role of mitochondrial reactive oxygen species and calciumChanges in mitochondrial dynamics during ceramide-induced cardiomyocyte early apoptosisExercise and Doxorubicin Treatment Modulate Cardiac Mitochondrial Quality Control Signaling.Heat shock protects cardiac cells from doxorubicin-induced toxicity by activating p38 MAPK and phosphorylation of small heat shock protein 27.Capillary electrophoresis monitors enhancement in subcellular reactive oxygen species production upon treatment with doxorubicin.Effects of doxorubicin on cardiac muscle subsarcolemmal and intermyofibrillar mitochondria.Cardiomyocyte specific ablation of p53 is not sufficient to block doxorubicin induced cardiac fibrosis and associated cytoskeletal changesShort-term exercise training protects against doxorubicin-induced cardiac mitochondrial damage independent of HSP72.Quercetin attenuates doxorubicin cardiotoxicity by modulating Bmi-1 expression.QiShenYiQi Pills, a Compound Chinese Medicine, Ameliorates Doxorubicin-Induced Myocardial Structure Damage and Cardiac Dysfunction in RatsIsorhamnetin protects against doxorubicin-induced cardiotoxicity in vivo and in vitro.Total Flavonoids from Clinopodium chinense (Benth.) O. Ktze Protect against Doxorubicin-Induced Cardiotoxicity In Vitro and In Vivo.Baicalein protects against doxorubicin-induced cardiotoxicity by attenuation of mitochondrial oxidant injury and JNK activation.Oral treatment with the herbal formula B307 alleviates cardiac toxicity in doxorubicin-treated mice via suppressing oxidative stress, inflammation, and apoptosisIn Vivo Protective Effects of Diosgenin against Doxorubicin-Induced Cardiotoxicity.Analysis of proteome changes in doxorubicin-treated adult rat cardiomyocyte.Role of exercise in rehabilitation of cancer survivors.Monitoring doxorubicin cellular uptake and trafficking using in vitro Raman microspectroscopy: short and long time exposure effects on lung cancer cell lines.Adriamycin-induced oxidative mitochondrial cardiotoxicity.A novel function for hydroxyproline oxidase in apoptosis through generation of reactive oxygen species.RNA interference-mediated FANCF silencing sensitizes OVCAR3 ovarian cancer cells to adriamycin through increased adriamycin-induced apoptosis dependent on JNK activation.Modeling Doxorubicin-Induced Cardiotoxicity in Human Pluripotent Stem Cell Derived-CardiomyocytesMolecular and cellular mechanisms of anthracycline cardiotoxicity.Hyperglycemic oxoaldehyde, glyoxal, causes barrier dysfunction, cytoskeletal alterations, and inhibition of angiogenesis in vascular endothelial cells: aminoguanidine protection.Akt3 knockdown induces mitochondrial dysfunction in human cancer cells.Beta-adrenergic receptor mediated protection against doxorubicin-induced apoptosis in cardiomyocytes: the impact of high ambient glucose.Acetaminophen attenuates doxorubicin-induced cardiac fibrosis via osteopontin and GATA4 regulation: reduction of oxidant levels.Poly(ADP-ribose) polymerase regulates myocardial calcium handling in doxorubicin-induced heart failureNeuregulin-1 attenuated doxorubicin-induced decrease in cardiac troponins.All-trans retinoic acid protects against doxorubicin-induced cardiotoxicity by activating the ERK2 signalling pathwayProtective effect of tetrahydroxystilbene glucoside on cardiotoxicity induced by doxorubicin in vitro and in vivo.Possibilities to increase the effectiveness of doxorubicin in cancer cells killing.Imaging of early modification in cardiomyopathy: the doxorubicin-induced model.Molecular basis of cancer-therapy-induced cardiotoxicity: introducing microRNA biomarkers for early assessment of subclinical myocardial injury.Oxidative stress does not play a primary role in the toxicity induced with clinical doses of doxorubicin in myocardial H9c2 cells.A Novel Danshensu Derivative Prevents Cardiac Dysfunction and Improves the Chemotherapeutic Efficacy of Doxorubicin in Breast Cancer Cells.Pathways of cardiac toxicity: comparison between chemotherapeutic drugs doxorubicin and mitoxantrone.Doxorubicin-Mediated Cardiotoxicity: Role of Mitochondrial Connexin 43.Effect of monochromatic visible light on intracellular superoxide anion production and mitochondrial membrane potential of B16F1 and B16F10 murine melanoma cells.
P2860
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P2860
Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis.
@en
type
label
Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis.
@en
prefLabel
Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis.
@en
P1476
Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis.
@en
P2093
Christiaan Leeuwenburgh
Pattie S Green
P304
P356
10.1016/S0925-4439(02)00144-8
P577
2002-10-01T00:00:00Z