about
The Innate Immune System in Acute and Chronic WoundsRobotic repair of uterine dehiscenceInterferon-inducible protein 9 (CXCL11)-induced cell motility in keratinocytes requires calcium flux-dependent activation of mu-calpain.Expression of fibroblast growth factor binding protein in head and neck cancer.Platelet-rich plasma (PRP): methodological aspects and clinical applications.Heat shock proteins modulate keloid formation.Delayed wound repair in sepsis is associated with reduced local pro-inflammatory cytokine expression.Effect of growth factors on antimicrobial peptides and pro-inflammatory mediators during wound healing.IP-10 inhibits epidermal growth factor-induced motility by decreasing epidermal growth factor receptor-mediated calpain activity.Platelet-rich plasma and platelet gel: a reviewSystems-based approaches toward wound healing.Experimental study of comparing rhEGF with rhβFGF on improving the quality of wound healing.The GH/IGF-1 system in critical illnessPlatelet-rich plasma and the elimination of neuropathic pain.Elevations in growth hormone and glucagon-like peptide-2 levels on admission are associated with increased mortality in trauma patients.Associations between levels of insulin-like growth factor 1 and sinusoidal obstruction syndrome after allogeneic haematopoietic stem cell transplantation.The use of growth factors and other humoral agents to accelerate and enhance burn wound healing.Aging fibroblasts present reduced epidermal growth factor (EGF) responsiveness due to preferential loss of EGF receptors.Exogenous liposomal IGF-I cDNA gene transfer leads to endogenous cellular and physiological responses in an acute wound.IGF-I gene transfer in thermally injured rats.Liposomal IGF-1 gene transfer modulates pro- and anti-inflammatory cytokine mRNA expression in the burn wound.Therapeutic success and efficacy of nonviral liposomal cDNA gene transfer to the skin in vivo is dose dependent.Designing the stem cell microenvironment for guided connective tissue regeneration.IGF-I gene transfer effects on inflammatory elements present after thermal trauma.Tight glycaemic control is a key factor in wound healing enhancement strategies in an experimental diabetes mellitus model.Stimulation of cutaneous wound healing by an FPR2-specific peptide agonist WKYMVm.Insulinlike growth factor I plus insulinlike growth factor binding protein 3 attenuates the proinflammatory acute phase response in severely burned children.
P2860
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P2860
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Growth factors and wound healing.
@en
type
label
Growth factors and wound healing.
@en
prefLabel
Growth factors and wound healing.
@en
P2860
P1476
Growth factors and wound healing.
@en
P2093
H H Steenfos
P2860
P304
P356
10.3109/02844319409071186
P577
1994-06-01T00:00:00Z