Spread of hepatitis B viruses in vitro requires extracellular progeny and may be codetermined by polarized egress. - Wikidata - wikimedia - TriplyDB

Spread of hepatitis B viruses in vitro requires extracellular progeny and may be codetermined by polarized egress.

Spread of hepatitis B viruses in vitro requires extracellular progeny and may be codetermined by polarized egress.

http://www.wikidata.org/entity/Q40700533

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Hepatitis B virus morphogenesis Viral and cellular determinants involved in hepadnaviral entry Identification of a structural motif crucial for infectivity of hepatitis B viruses Proteoglycans act as cellular hepatitis delta virus attachment receptors.Hepatocytes traffic and export hepatitis B virus basolaterally by polarity-dependent mechanisms Modelling the Impact of Cell-To-Cell Transmission in Hepatitis B Virus.Is hepatitis B-virucidal validation of biocides possible with the use of surrogates?Hepatitis B virus infection initiates with a large surface protein-dependent binding to heparan sulfate proteoglycans.Duck hepatitis B virus requires cholesterol for endosomal escape during virus entry Human stem cell-derived hepatocytes as a model for hepatitis B virus infection, spreading and virus-host interactions.Hepatitis B virus: virology, molecular biology, life cycle and intrahepatic spread.Acute HBV infection in humanized chimeric mice has multiphasic viral kinetics.

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P2860

Spread of hepatitis B viruses in vitro requires extracellular progeny and may be codetermined by polarized egress.

http://www.wikidata.org/entity/Q40700533

description

2004 nî lūn-bûn

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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2004年论文

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2004年论文

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name

Spread of hepatitis B viruses ...... etermined by polarized egress.

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Spread of hepatitis B viruses ...... etermined by polarized egress.

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Spread of hepatitis B viruses ...... etermined by polarized egress.

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JVI.78.8.3977-3983.2004

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Journal of Virology

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Spread of hepatitis B viruses ...... etermined by polarized egress.

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P2093

Funk A

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Hohenberg H

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Mhamdi M

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Sirma H

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Will H

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P2860

Residues critical for duck hepatitis B virus neutralization are involved in host cell interaction.

Directed egress of animal viruses promotes cell-to-cell spread

The A34R glycoprotein gene is required for induction of specialized actin-containing microvilli and efficient cell-to-cell transmission of vaccinia virus

Herpes simplex virus glycoproteins E and I facilitate cell-to-cell spread in vivo and across junctions of cultured cells.

New hepatitis B virus of cranes that has an unexpected broad host range.

Suramin prevents duck hepatitis B virus infection in vivo.

Uptake of duck hepatitis B virus into hepatocytes occurs by endocytosis but does not require passage of the virus through an acidic intracellular compartment.

Antibody-sensitive and antibody-resistant cell-to-cell spread by vaccinia virus: role of the A33R protein in antibody-resistant spread.

Suramin inhibits in vitro infection by duck hepatitis B virus, Rous sarcoma virus, and hepatitis delta virus.

Mechanism of HIV spread from lymphocytes to epithelia.

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3977-3983

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scholarly article

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10.1128/JVI.78.8.3977-3983.2004

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374299

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