TAT protein transduction into isolated perfused hearts: TAT-apoptosis repressor with caspase recruitment domain is cardioprotective.
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Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activationThe apoptosis inhibitor ARC undergoes ubiquitin-proteasomal-mediated degradation in response to death stimuli: identification of a degradation-resistant mutantJuvenile exposure to anthracyclines impairs cardiac progenitor cell function and vascularization resulting in greater susceptibility to stress-induced myocardial injury in adult miceDelivery of nucleic acids and nanomaterials by cell-penetrating peptides: opportunities and challengesBeta-actin association with endothelial nitric-oxide synthase modulates nitric oxide and superoxide generation from the enzymeTranscription factor Foxo3a prevents apoptosis by regulating calcium through the apoptosis repressor with caspase recruitment domainARC protects rat cardiomyocytes against oxidative stress through inhibition of caspase-2 mediated mitochondrial pathwayProtection of cardiomyocytes from ischemic/hypoxic cell death via Drbp1 and pMe2GlyDH in cardio-specific ARC transgenic miceIn vivo protein transduction: delivery of PEP-1-SOD1 fusion protein into myocardium efficiently protects against ischemic insult.Autophagic program is regulated by miR-325The role of apoptosis repressor with a CARD domain (ARC) in the therapeutic resistance of renal cell carcinoma (RCC): the crucial role of ARC in the inhibition of extrinsic and intrinsic apoptotic signalling.Xenotransplantation of mitochondrial electron transfer enzyme, Ndi1, in myocardial reperfusion injury.Sex-related resistance to myocardial ischemia-reperfusion injury is associated with high constitutive ARC expressionAutophagy induced by ischemic preconditioning is essential for cardioprotection.Antibodies against Marinobacter algicola and Salmonella typhimurium flagellins do not cross-neutralize TLR5 activationCardiomyopathy of Friedreich's ataxia: use of mouse models to understand human disease and guide therapeutic development.The Heart in Friedreich's Ataxia: Basic Findings and Clinical Implications.HIV Tat, its TARgets and the control of viral gene expression.Biodistribution and blood clearance of plasmid DNA administered in arginine peptide complexes.Cell-permeable protein therapy for complex I dysfunctionDon't lose heart--therapeutic value of apoptosis prevention in the treatment of cardiovascular disease.Recent advances in the use of protein transduction domains for the delivery of peptides, proteins and nucleic acids in vivo.Transactivating transcriptional activator-mediated drug delivery.Programmed cell death in cardiac myocytes: strategies to maximize post-ischemic salvage.The rationale for cardiomyocyte resuscitation in myocardial salvage.TGF-β improves myocardial function and prevents apoptosis induced by anoxia-reoxygenation, through the reduction of endoplasmic reticulum stress.Knockdown of apoptosis repressor with caspase recruitment domain (ARC) increases the sensitivity of human glioma cell line U251MG to VM-26.Inhibition of coxsackievirus B3 in cell cultures and in mice by peptide-conjugated morpholino oligomers targeting the internal ribosome entry site.The heat shock protein 27 (Hsp27) operates predominantly by blocking the mitochondrial-independent/extrinsic pathway of cellular apoptosis.Necrostatin-1 protects against glutamate-induced glutathione depletion and caspase-independent cell death in HT-22 cells.Can ARC save the heart?Regulation of necrosis of H9c2 myogenic cells upon transient energy deprivation. Rapid deenergization of mitochondria precedes necrosis and is controlled by reactive oxygen species, stress kinase JNK, HSP72 and ARC.From gene expression profiles to biological validation in enteroviral heart disease.The apoptosis repressor with a CARD domain (ARC) gene is a direct hypoxia-inducible factor 1 target gene and promotes survival and proliferation of VHL-deficient renal cancer cells.Autophagy is required for preconditioning by the adenosine A1 receptor-selective agonist CCPA.Small G-protein Rho is involved in the maintenance of cardiac myocyte morphology.Enhancement of gene targeting in human cells by intranuclear permeation of the Saccharomyces cerevisiae Rad52 proteinUbiquitination and degradation of the anti-apoptotic protein ARC by MDM2.The cardioprotective effect of postconditioning is mediated by ARC through inhibiting mitochondrial apoptotic pathway.Nicotine plus a high-fat diet triggers cardiomyocyte apoptosis.
P2860
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P2860
TAT protein transduction into isolated perfused hearts: TAT-apoptosis repressor with caspase recruitment domain is cardioprotective.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
TAT protein transduction into ...... nt domain is cardioprotective.
@en
type
label
TAT protein transduction into ...... nt domain is cardioprotective.
@en
prefLabel
TAT protein transduction into ...... nt domain is cardioprotective.
@en
P2093
P1433
P1476
TAT protein transduction into ...... nt domain is cardioprotective.
@en
P2093
Asa B Gustafsson
M Richard Sayen
Michael T Crow
Roberta A Gottlieb
Scott D Williams
P304
P356
10.1161/01.CIR.0000023943.50821.F7
P407
P577
2002-08-01T00:00:00Z