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Tick-Host-Pathogen Interactions: Conflict and CooperationComparative Proteomics Identifies Host Immune System Proteins Affected by Infection with Mycobacterium bovisIdentification and Characterization of Anaplasma phagocytophilum Proteins Involved in Infection of the Tick Vector, Ixodes scapularisOral vaccination with heat inactivated Mycobacterium bovis activates the complement system to protect against tuberculosisEhrlichia minasensis sp. nov., a new species within the genus Ehrlichia isolated from the tick Rhipicephalus microplus.Tick-host conflict: immunoglobulin E antibodies to tick proteins in patients with anaphylaxis to tick biteTick cell culture isolation and growth of Rickettsia raoultii from Dutch Dermacentor reticulatus ticksReciprocal regulation of NF-kB (Relish) and Subolesin in the tick vector, Ixodes scapularis.A novel Lawsonia intracellularis autotransporter protein is a prominent antigen.Systems biology of tissue-specific response to Anaplasma phagocytophilum reveals differentiated apoptosis in the tick vector Ixodes scapularis.Nuclease Tudor-SN Is Involved in Tick dsRNA-Mediated RNA Interference and Feeding but Not in Defense against Flaviviral or Anaplasma phagocytophilum Rickettsial InfectionDetection and identification of putative bacterial endosymbionts and endogenous viruses in tick cell lines.Tissue-Specific Signatures in the Transcriptional Response to Anaplasma phagocytophilum Infection of Ixodes scapularis and Ixodes ricinus Tick Cell LinesGene silencing in tick cell lines using small interfering or long double-stranded RNA.Integrated Metabolomics, Transcriptomics and Proteomics Identifies Metabolic Pathways Affected by Anaplasma phagocytophilum Infection in Tick Cells.Anaplasma phagocytophilum increases the levels of histone modifying enzymes to inhibit cell apoptosis and facilitate pathogen infection in the tick vector Ixodes scapularis.Anaplasma phagocytophilum inhibits apoptosis and promotes cytoskeleton rearrangement for infection of tick cellsTick vaccines and the control of tick-borne pathogens.The intracellular bacterium Anaplasma phagocytophilum selectively manipulates the levels of vertebrate host proteins in the tick vector Ixodes scapularisTick capillary feeding for the study of proteins involved in tick-pathogen interactions as potential antigens for the control of tick infestation and pathogen infectionAnaplasma phagocytophilum Infection Subverts Carbohydrate Metabolic Pathways in the Tick Vector, Ixodes scapularis.Effect of blood type on anti-α-Gal immunity and the incidence of infectious diseases.Tick-Pathogen Interactions and Vector Competence: Identification of Molecular Drivers for Tick-Borne Diseases.Ixodes scapularis Tick Cells Control Anaplasma phagocytophilum Infection by Increasing the Synthesis of Phosphoenolpyruvate from Tyrosine.Vaccinomics Approach to the Identification of Candidate Protective Antigens for the Control of Tick Vector Infestations and Anaplasma phagocytophilum InfectionRemodeling of tick cytoskeleton in response to infection with Anaplasma phagocytophilum.Infection of Ixodes spp. tick cells with different Anaplasma phagocytophilum isolates induces the inhibition of apoptotic cell death.Field evidence that roe deer (Capreolus capreolus) are a natural host for Ehrlichia phagocytophila.Coinfection of tick cell lines has variable effects on replication of intracellular bacterial and viral pathogens.Anaplasma phagocytophilum MSP4 and HSP70 Proteins Are Involved in Interactions with Host Cells during Pathogen Infection.Evidence of co-infection with Mycobacterium bovis and tick-borne pathogens in a naturally infected sheep flock.Vaccination against Louping Ill Virus Protects Goats from Experimental Challenge with Spanish Goat Encephalitis Virus.Vaccinomics Approach to Tick Vaccine Development.Identification and characterization of a novel tick-borne flavivirus subtype in goats (Capra hircus) in Spain.Expression by Lawsonia intracellularis of type III secretion system components during infection.Proteomic analysis of Lawsonia intracellularis reveals expression of outer membrane proteins during infection.Anaplasma phagocytophilum Manipulates Host Cell Apoptosis by Different Mechanisms to Establish Infection.Draft Genome Sequences of Anaplasma phagocytophilum, A. marginale, and A. ovis Isolates from Different Hosts.Oral administration of heat-inactivated Mycobacterium bovis reduces the response of farmed red deer to avian and bovine tuberculin.Tick galactosyltransferases are involved in α-Gal synthesis and play a role during Anaplasma phagocytophilum infection and Ixodes scapularis tick vector development
P50
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P50
description
hulumtuese
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onderzoeker
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researcher
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հետազոտող
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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M Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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Pilar Alberdi
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P1053
K-7788-2014
P106
P1153
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P2038
Pilar_Alberdi
P21
P2798
P31
P3829
P496
0000-0002-6999-9735
P569
2000-01-01T00:00:00Z