SipA, SopA, SopB, SopD, and SopE2 contribute to Salmonella enterica serotype typhimurium invasion of epithelial cells.
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Caveolin-1 mediates Salmonella invasion via the regulation of SopE-dependent Rac1 activation and actin reorganizationSalmonella takes control: effector-driven manipulation of the hostA Salmonella small non-coding RNA facilitates bacterial invasion and intracellular replication by modulating the expression of virulence factorsThe inflammation-associated Salmonella SopA is a HECT-like E3 ubiquitin ligaseThe frequency and duration of Salmonella-macrophage adhesion events determines infection efficiencyTranscription modulation of Salmonella enterica serovar Typhimurium promoters by sub-MIC levels of rifampinThe PagN protein of Salmonella enterica serovar Typhimurium is an adhesin and invasin.The SdiA-regulated gene srgE encodes a type III secreted effector.S. Typhimurium sseJ gene decreases the S. Typhi cytotoxicity toward cultured epithelial cells.Analysis of interactions of Salmonella type three secretion mutants with 3-D intestinal epithelial cellsGenomic characterization of Salmonella Cerro ST367, an emerging Salmonella subtype in cattle in the United States.Infection of mice by Salmonella enterica serovar Enteritidis involves additional genes that are absent in the genome of serovar Typhimurium.The inflammatory cytokine tumor necrosis factor modulates the expression of Salmonella typhimurium effector proteins.Deciphering why Salmonella Gallinarum is less invasive in vitro than Salmonella EnteritidisComplete proteome of a quinolone-resistant Salmonella Typhimurium phage type DT104B clinical strain.Differential phenotypic diversity among epidemic-spanning Salmonella enterica serovar enteritidis isolates from humans or animals.Virulence of broad- and narrow-host-range Salmonella enterica serovars in the streptomycin-pretreated mouse model.Glycerol supplementation enhances L. reuteri's protective effect against S. Typhimurium colonization in a 3-D model of colonic epithelium.Antimicrobial resistance, virulence-associated genes, and pulsed-field gel electrophoresis profiles of Salmonella enterica subsp. enterica serovar Typhimurium isolated from piglets with diarrhea in KoreaExploitation of the ubiquitin system by invading bacteriaA small non-coding RNA of the invasion gene island (SPI-1) represses outer membrane protein synthesis from the Salmonella core genome.Role of Salmonella Pathogenicity Island 1 protein IacP in Salmonella enterica serovar typhimurium pathogenesis.Novel determinants of intestinal colonization of Salmonella enterica serotype typhimurium identified in bovine enteric infection.Establishment of systemic Brucella melitensis infection through the digestive tract requires urease, the type IV secretion system, and lipopolysaccharide O antigenSalmonella - at home in the host cell.Analysis of the Salmonella regulatory network suggests involvement of SsrB and H-NS in σ(E)-regulated SPI-2 gene expressionSalmonella Interaction with and Passage through the Intestinal Mucosa: Through the Lens of the OrganismA Salmonella type three secretion effector/chaperone complex adopts a hexameric ring-like structure.Selected lactic acid-producing bacterial isolates with the capacity to reduce Salmonella translocation and virulence gene expression in chickens.Differences in Salmonella enterica serovar Typhimurium strain invasiveness are associated with heterogeneity in SPI-1 gene expressionImpairment of swimming motility by antidiarrheic Lactobacillus acidophilus strain LB retards internalization of Salmonella enterica serovar Typhimurium within human enterocyte-like cells.Non-typhoidal Salmonella Typhimurium ST313 isolates that cause bacteremia in humans stimulate less inflammasome activation than ST19 isolates associated with gastroenteritisA Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.Genomic Comparison of Non-Typhoidal Salmonella enterica Serovars Typhimurium, Enteritidis, Heidelberg, Hadar and Kentucky Isolates from Broiler Chickens.Salmonella, the host and its microbiota.Salmonella effectors: important players modulating host cell function during infection.Capsule-mediated immune evasion: a new hypothesis explaining aspects of typhoid fever pathogenesisSalmonella enterica serovar Senftenberg human clinical isolates lacking SPI-1.Campylobacter jejuni CsrA mediates oxidative stress responses, biofilm formation, and host cell invasion.Role for myosin II in regulating positioning of Salmonella-containing vacuoles and intracellular replication.
P2860
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P2860
SipA, SopA, SopB, SopD, and SopE2 contribute to Salmonella enterica serotype typhimurium invasion of epithelial cells.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
SipA, SopA, SopB, SopD, and So ...... invasion of epithelial cells.
@en
type
label
SipA, SopA, SopB, SopD, and So ...... invasion of epithelial cells.
@en
prefLabel
SipA, SopA, SopB, SopD, and So ...... invasion of epithelial cells.
@en
P2093
P2860
P50
P1476
SipA, SopA, SopB, SopD, and So ...... m invasion of epithelial cells
@en
P2093
Helene Andrews-Polymenis
L Garry Adams
Quynh T Tran
R Paul Wilson
Sangeeta Khare
Sara Lawhon
P2860
P304
P356
10.1128/IAI.73.1.146-154.2005
P407
P577
2005-01-01T00:00:00Z