Trans-splicing in Higher Eukaryotes: Implications for Cancer Development?
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Evolutionary Insights into RNA trans-Splicing in VertebratesHypothesis: Artifacts, Including Spurious Chimeric RNAs with a Short Homologous Sequence, Caused by Consecutive Reverse Transcriptions and Endogenous Random PrimersChimeric RNAs in cancer and normal physiology.Intergenically Spliced Chimeric RNAs in Cancer.Recurrent chimeric fusion RNAs in non-cancer tissues and cells.It Is Imperative to Establish a Pellucid Definition of Chimeric RNA and to Clear Up a Lot of Confusion in the Relevant Research.Do non-genomically encoded fusion transcripts cause recurrent chromosomal translocations?Evolving techniques for gene fusion detection in soft tissue tumours.The emerging complexity of gene fusions in cancer.Novel fusion genes and chimeric transcripts in ependymal tumors.The role of canonical and noncanonical pre-mRNA splicing in plant stress responses.Atypical RNAs in the coelacanth transcriptome.Transcriptional-Readthrough RNAs Reflect the Phenomenon of "A Gene Contains Gene(s)" or "Gene(s) within a Gene" in the Human Genome, and Thus Are Not Chimeric RNAs.
P2860
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P2860
Trans-splicing in Higher Eukaryotes: Implications for Cancer Development?
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Trans-splicing in Higher Eukaryotes: Implications for Cancer Development?
@en
type
label
Trans-splicing in Higher Eukaryotes: Implications for Cancer Development?
@en
prefLabel
Trans-splicing in Higher Eukaryotes: Implications for Cancer Development?
@en
P2860
P356
P1476
Trans-splicing in Higher Eukaryotes: Implications for Cancer Development?
@en
P2860
P356
10.3389/FGENE.2011.00092
P577
2011-12-26T00:00:00Z