Diets rich in fructose, fat or fructose and fat alter intestinal barrier function and lead to the development of nonalcoholic fatty liver disease over time.
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High Dietary Fructose: Direct or Indirect Dangerous Factors Disturbing Tissue and Organ FunctionsOral arginine supplementation protects female mice from the onset of non-alcoholic steatohepatitis.Toxic AGE (TAGE) Theory for the Pathophysiology of the Onset/Progression of NAFLD and ALDExposure to bacterial products lipopolysaccharide and flagellin and hepatocellular carcinoma: a nested case-control study.Insulin Production and Resistance in Different Models of Diet-Induced Obesity and Metabolic Syndrome.Treatment with alpha-galactosylceramide protects mice from early onset of nonalcoholic steatohepatitis: Role of intestinal barrier function.Fructose and NAFLD: The Multifaceted Aspects of Fructose Metabolism.Nonalcoholic Fatty Liver Disease, the Gut Microbiome, and Diet.Oral citrulline supplementation protects female mice from the development of non-alcoholic fatty liver disease (NAFLD).Dietary fructose causes defective insulin signalling and ceramide accumulation in the liver that can be reversed by gut microbiota modulationFructose: A Dietary Sugar in Crosstalk with Microbiota Contributing to the Development and Progression of Non-Alcoholic Liver DiseaseChronic consumption of fructose in combination with trans fatty acids but not with saturated fatty acids induces nonalcoholic steatohepatitis with fibrosis in rats.Short-Term Intake of a Fructose-, Fat- and Cholesterol-Rich Diet Causes Hepatic Steatosis in Mice: Effect of Antibiotic Treatment.Preventive effects of dietary walnuts on high-fat-induced hepatic fat accumulation, oxidative stress and apoptosis in mice.Animal Models of Nonalcoholic Fatty Liver Disease-A Starter's Guide.Recent advances in biological production of erythritol.fat-1 mice prevent high-fat plus high-sugar diet-induced non-alcoholic fatty liver disease.Diet high in fructose promotes liver steatosis and hepatocyte apoptosis in C57BL/6J female mice: Role of disturbed lipid homeostasis and increased oxidative stress.Natural products, an important resource for discovery of multitarget drugs and functional food for regulation of hepatic glucose metabolism.Loss of lipopolysaccharide-binding protein attenuates the development of diet-induced non-alcoholic fatty liver disease in mice.Oral administration of fructose exacerbates liver fibrosis and hepatocarcinogenesis via increased intestinal permeability in a rat steatohepatitis model.Colon Epithelial MicroRNA Network in Fatty LiverNon-Alcoholic Fatty Liver Disease in Overweight Children: Role of Fructose Intake and Dietary Pattern
P2860
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P2860
Diets rich in fructose, fat or fructose and fat alter intestinal barrier function and lead to the development of nonalcoholic fatty liver disease over time.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Diets rich in fructose, fat or ...... fatty liver disease over time.
@en
type
label
Diets rich in fructose, fat or ...... fatty liver disease over time.
@en
prefLabel
Diets rich in fructose, fat or ...... fatty liver disease over time.
@en
P2093
P1476
Diets rich in fructose, fat or ...... fatty liver disease over time.
@en
P2093
Anna Janina Engstler
Astrid Spruss
Cathrin Sellmann
Cheng Jun Jin
Christian Degen
Ina Bergheim
Josephine Priebs
Marianne Landmann
Otmar Huber
Stefanie Gärttner
P304
P356
10.1016/J.JNUTBIO.2015.05.011
P577
2015-06-16T00:00:00Z