Contribution of the ADP-ribosylating and receptor-binding properties of cholera-like enterotoxins in modulating cytokine secretion by human intestinal epithelial cells.
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The B subunit of an AB5 toxin produced by Salmonella enterica serovar Typhi up-regulates chemokines, cytokines, and adhesion molecules in human macrophage, colonic epithelial, and brain microvascular endothelial cell linesEscherichia coli heat-labile detoxified enterotoxin modulates dendritic cell function and attenuates allergic airway inflammationProphylactic administration of bacterially derived immunomodulators improves the outcome of influenza virus infection in a murine model.Probing local innate immune responses after mucosal immunisationThe A subunit of Escherichia coli heat-labile enterotoxin functions as a mucosal adjuvant and promotes IgG2a, IgA, and Th17 responses to vaccine antigensSimultaneous exposure to Escherichia coli heat-labile and heat-stable enterotoxins increases fluid secretion and alters cyclic nucleotide and cytokine production by intestinal epithelial cellsMutant Escherichia coli heat-labile toxin B subunit that separates toxoid-mediated signaling and immunomodulatory action from trafficking and delivery functionsContext-dependent activation kinetics elicited by soluble versus outer membrane vesicle-associated heat-labile enterotoxin.Exploiting immune surveillance mechanisms in mucosal vaccine development.ADP-ribosylating bacterial enzymes for the targeted control of mucosal tolerance and immunity.Cholera toxin, E. coli heat-labile toxin, and non-toxic derivatives induce dendritic cell migration into the follicle-associated epithelium of Peyer's patches.The chromosomal nature of LT-II enterotoxins solved: a lambdoid prophage encodes both LT-II and one of two novel pertussis-toxin-like toxin family members in type II enterotoxigenic Escherichia coli.Mechanisms of Cholera Toxin in the Modulation of TH17 Responses.Mucosal adjuvants and long-term memory development with special focus on CTA1-DD and other ADP-ribosylating toxins.Transcriptional upregulation of inflammatory cytokines in human intestinal epithelial cells following Vibrio cholerae infection.Cholera toxin induces a transient depletion of CD8+ intraepithelial lymphocytes in the rat small intestine as detected by microarray and immunohistochemistry.Bacterial exotoxins downregulate cathelicidin (hCAP-18/LL-37) and human beta-defensin 1 (HBD-1) expression in the intestinal epithelial cells.
P2860
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P2860
Contribution of the ADP-ribosylating and receptor-binding properties of cholera-like enterotoxins in modulating cytokine secretion by human intestinal epithelial cells.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Contribution of the ADP-ribosy ...... n intestinal epithelial cells.
@en
type
label
Contribution of the ADP-ribosy ...... n intestinal epithelial cells.
@en
prefLabel
Contribution of the ADP-ribosy ...... n intestinal epithelial cells.
@en
P2093
P1433
P1476
Contribution of the ADP-ribosy ...... n intestinal epithelial cells.
@en
P2093
Lorna Bailey
Marco Soriani
Timothy R Hirst
P304
P356
10.1099/00221287-148-3-667
P577
2002-03-01T00:00:00Z