Novel pathways associated with bypassing cellular senescence in human prostate epithelial cells.
about
Therapeutic targeting of replicative immortalityEGF-mediated regulation of IGFBP-3 determines esophageal epithelial cellular response to IGF-IThe senescence-associated secretory phenotype: the dark side of tumor suppressionRegulation of phosphatase homologue of tensin protein expression by bone morphogenetic proteins in prostate epithelial cells.Senescent keratinocytes die by autophagic programmed cell death.Comparative analysis of gene expression data reveals novel targets of senescence-associated microRNAs.Senescent vs. non-senescent cells in the human annulus in vivo: cell harvest with laser capture microdissection and gene expression studies with microarray analysis.Epigenetic mechanisms of promigratory chemokine CXCL14 regulation in human prostate cancer cells.Transcriptional networks and cellular senescence in human mammary fibroblasts.Epidermal growth factor receptor regulates aberrant expression of insulin-like growth factor-binding protein 3IGFBP-3, a marker of cellular senescence, is overexpressed in human papillomavirus-immortalized cervical cells and enhances IGF-1-induced mitogenesisIL-1 induces IGF-dependent epithelial proliferation in prostate development and reactive hyperplasiaTherapy-induced senescence in cancerInsights into gene expression changes impacting B-cell transformation: cross-species microarray analysis of bovine leukemia virus tax-responsive genes in ovine B cellsSensitivity to TOP2 targeting chemotherapeutics is regulated by Oct1 and FILIP1LThe expression signature of in vitro senescence resembles mouse but not human aging.Regulation of senescence in cancer and aging.The identification of senescence-specific genes during the induction of senescence in prostate cancer cells.Senescence-specific gene expression fingerprints reveal cell-type-dependent physical clustering of up-regulated chromosomal loci.Down-regulation of Filamin A interacting protein 1-like Is associated with promoter methylation and an invasive phenotype in breast, colon, lung and pancreatic cancers [corrected]IGFBP-3 is a metastasis suppression gene in prostate cancerDownregulation of Filamin A interacting protein 1-like is associated with promoter methylation and induces an invasive phenotype in ovarian cancer.Smurf2-mediated ubiquitination and degradation of Id1 regulates p16 expression during senescence.Cellular senescence and organismal aging.The tumor suppressor ING1b is a novel corepressor for the androgen receptor and induces cellular senescence in prostate cancer cells.Decreased skp2 expression is necessary but not sufficient for therapy-induced senescence in prostate cancerDrug-induced senescence bystander proliferation in prostate cancer cells in vitro and in vivo.Functional characterization of filamin a interacting protein 1-like, a novel candidate for antivascular cancer therapy.Critical pathways in cellular senescence and immortalization revealed by gene expression profiling.Recent insights into the molecular mechanisms involved in aging and the malignant transformation of adult stem/progenitor cells and their therapeutic implications.Androgen control of cell proliferation and cytoskeletal reorganization in human fibrosarcoma cells: role of RhoB signaling.Perspectives in mammalian IGFBP-3 biology: local vs. systemic action.Insulin-like growth factor binding protein-3 has dual effects on gastrointestinal stromal tumor cell viability and sensitivity to the anti-tumor effects of imatinib mesylate in vitro.Resveratrol-induced gene expression profiles in human prostate cancer cells.Filamin A interacting protein 1-like inhibits WNT signaling and MMP expression to suppress cancer cell invasion and metastasis.Silencing of insulin-like growth factor-binding protein-2 in human glioblastoma cells reduces both invasiveness and expression of progression-associated gene CD24.Global effects of human papillomavirus type 18 E6/E7 in an organotypic keratinocyte culture system.Low p16INK4a Expression in Early Passage Human Prostate Basal Epithelial Cells Enables Immortalization by Telomerase Expression Alone.Truncation of inhibitor of growth family protein 5 effectively induces senescence, but not apoptosis in human tongue squamous cell carcinoma cell line.Altered apoptotic responses in neurons lacking RhoB GTPase.
P2860
Q27027457-44F91B25-1774-440F-B5C0-86E0351AF3ACQ28275900-335F1F66-F4B7-4924-B309-1BA8DA70CEBEQ29620106-E221D414-08C3-46AC-8CB9-59F4A76CB069Q30426736-4D8F2FDF-4BAD-464A-B381-8A07B8D85F70Q30485600-1CA5437F-6A31-423E-B1B0-0406164BF975Q30829491-E61C8971-E6A4-413B-BF29-D16DF86229ADQ33527140-76AC3925-D11F-462A-A184-0C81DE12DD15Q33551404-AAE70955-05D8-493C-BFBF-5074DDC9CA26Q33763939-DA050A5C-B17F-4670-A1F7-199C58E31F22Q34073234-5F337B59-A1AA-4F2D-8F16-F9FD0135E85FQ34151999-0D719EB9-892A-414B-A0C0-348B55994541Q34171551-5153823A-C302-41A3-B09E-AF86FE8E36A4Q34212773-B3428EC9-3929-4B35-8034-AB4FAC9B0A14Q34353406-95E144B8-CC0B-4FE0-86BA-6FCBE323844CQ34382668-6B43BCD9-D40C-4916-BD4A-B98CEAC8CEA5Q34481078-AC8513D0-7E57-4629-AC7C-542AACE4C102Q34665706-7E95CD43-FD87-498D-91B7-5AA7376ED48DQ34769742-306D23F6-F951-458E-8380-424834C66A13Q34868217-9AC85859-B948-4334-8A4C-B41E41F71C9EQ35067854-172C3D5D-51CC-4054-871E-1EFEB8162CF9Q35141734-3C14377B-53CC-4A65-8886-4422714C1247Q35169206-FC092C2A-D6DB-4E45-8921-AC455E1EEE64Q35549516-53214D7E-85ED-4EDB-A915-776089367A2DQ35815320-1B08C115-65C1-4E57-8D7C-42A10E4970A1Q35963298-5E2D0C38-3C5B-4DEF-99E2-D757582ED884Q36199857-8CB37CB0-6D36-4364-9863-3C4C3B98D37DQ36608493-DAF536B5-0217-403C-AD85-B056EF391638Q37038009-1885997B-BA00-44CE-91C2-A6B00949A045Q37246647-B70C2645-055F-4740-B6FC-F5841E7A3282Q37310063-4FD78A71-17E4-4EBD-B3D3-994853EDB98EQ37327005-8B685D7C-53F2-4095-BCB1-37E75D643117Q37412844-81B2E7A5-9F6B-4951-BB9D-7BF4B5B25581Q37428196-F851DAB7-5559-4574-8C91-5E06F8B7C408Q37467489-48F414E7-E00A-484F-B1B9-C18051C902E9Q37710265-4AFC4ACC-4B17-4A56-8ACE-1867C89A16C1Q38302138-045C67C9-5DA7-4450-BADE-FE74DB032F6CQ38337784-5A3B2444-8DF9-4F27-A3CD-824F5D8FE460Q38730507-5B5317BA-B07D-4952-A31B-B83999457E34Q39061155-CB4A1D9D-C99B-45FC-84D4-C807434DD4F8Q39205118-DD03E9C9-BC49-48F3-8513-F9662E3D9454
P2860
Novel pathways associated with bypassing cellular senescence in human prostate epithelial cells.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Novel pathways associated with ...... man prostate epithelial cells.
@en
type
label
Novel pathways associated with ...... man prostate epithelial cells.
@en
prefLabel
Novel pathways associated with ...... man prostate epithelial cells.
@en
P2093
P2860
P356
P1476
Novel pathways associated with ...... man prostate epithelial cells.
@en
P2093
David F Jarrard
James D Brooks
Lisa M Grabert
Samuel E DePrimo
Steven R Schwarze
Vivian X Fu
P2860
P304
14877-14883
P356
10.1074/JBC.M200373200
P407
P577
2002-02-08T00:00:00Z