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Mechanisms of lead-induced hypertension and cardiovascular diseaseInhibition of protein kinase C signaling protects prefrontal cortex dendritic spines and cognition from the effects of chronic stress.Prospective study of blood and tibia lead in women undergoing surgical menopause.Reduction of sperm motility in a male laboratory worker exposed to solvents: a case studyA structural insight into lead neurotoxicity and calmodulin activation by heavy metalsDevelopmental neurotoxicity: some old and new issuesHeavy metal toxicity and the environmentChemically diverse toxicants converge on Fyn and c-Cbl to disrupt precursor cell function.Chronic Kidney Disease and Exposure to Nephrotoxic MetalsToxicology of choroid plexus: special reference to metal-induced neurotoxicities.Probing a homoleptic PbS3 coordination environment in a designed peptide using 207Pb NMR spectroscopy: implications for understanding the molecular basis of lead toxicity.Effects of micronutrients on metal toxicity.Impact of developmental lead exposure on splenic factorsThe importance of stereochemically active lone pairs for influencing Pb(II) and As(III) protein bindingLow-level lead exposure triggers neuronal apoptosis in the developing mouse brainChildhood Blood Lead Levels and Symptoms of Attention Deficit Hyperactivity Disorder (ADHD): A Cross-Sectional Study of Mexican Children.Lead exposure promotes translocation of protein kinase C activities in rat choroid plexus in vitro, but not in vivo.Exome-wide association study identifies genetic polymorphisms of C12orf51, MYL2, and ALDH2 associated with blood lead levels in the general Korean population.pH-dependent coordination of Pb2+ to metallothionein2: structures and insight into lead detoxificationMolecular neurobiology of lead (Pb(2+)): effects on synaptic function.Regulation of inducible nitric oxide synthase expression in beta cells by environmental factors: heavy metals.Phosphorylation of membrane proteins in erythrocytes treated with lead.Lead poisoning: acute exposure of the heart to lead ions promotes changes in cardiac function and Cav1.2 ion channels.Lead exposure may affect gingival health in children.Lead Exposure and Oxidative Stress-A Life Course Approach in U.S. Adults
P2860
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P2860
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Lead-calcium interactions in cellular lead toxicity.
@en
type
label
Lead-calcium interactions in cellular lead toxicity.
@en
prefLabel
Lead-calcium interactions in cellular lead toxicity.
@en
P1433
P1476
Lead-calcium interactions in cellular lead toxicity
@en
P2093
P577
1993-06-01T00:00:00Z