Neurotoxicity of 24-hydroxycholesterol, an important cholesterol elimination product of the brain, may be prevented by vitamin E and estradiol-17beta.
about
ABCA1 and scavenger receptor class B, type I, are modulators of reverse sterol transport at an in vitro blood-brain barrier constituted of porcine brain capillary endothelial cellsRole of necroptosis in the pathogenesis of solid organ injuryOxidized cholesterol as the driving force behind the development of Alzheimer's diseasePolymorphism in the cholesterol 24S-hydroxylase gene is associated with Alzheimer's diseaseLoading into nanoparticles improves quercetin's efficacy in preventing neuroinflammation induced by oxysterolsDetermination of 24S- and 27-hydroxycholesterol in plasma by high-performance liquid chromatography-mass spectrometry.Differential effects of the estrogen receptor agonist estradiol on toxicity induced by enzymatically-derived or autoxidation-derived oxysterols in human ARPE-19 cells.The influence of age of lead exposure on adult gray matter volume.Modulation of cholesterol in midlife affords cognitive advantage during ageing - a role for altered redox balanceA novel potent synthetic steroidal liver X receptor agonist lowers plasma cholesterol and triglycerides and reduces atherosclerosis in LDLR(-/-) miceQuantitative detection of free 24S-hydroxycholesterol, and 27-hydroxycholesterol from human serumCholesterol: from heart attacks to Alzheimer's disease.Genetics, lifestyle and the roles of amyloid beta and oxidative stress in Alzheimer's disease.Membrane-targeted strategies for modulating APP and Abeta-mediated toxicity.Antioxidant modulation in restoring mitochondrial function in neurodegeneration.The enzyme lecithin-cholesterol acyltransferase esterifies cerebrosterol and limits the toxic effect of this oxysterol on SH-SY5Y cells.24(S)-hydroxycholesterol is actively eliminated from neuronal cells by ABCA1.Interaction between 24-hydroxycholesterol, oxidative stress, and amyloid-β in amplifying neuronal damage in Alzheimer's disease: three partners in crime.Effects of cholesterol and its 24S-OH and 25-OH oxysterols on choline acetyltransferase-positive neurons in brain slices.Association of CYP46 intron 2 polymorphism in Finnish Alzheimer's disease samples and a global scale summary.25-hydroxycholesterol induces mitochondria-dependent apoptosis via activation of glycogen synthase kinase-3beta in PC12 cells.The level of 24-Hydroxycholesteryl Esters is an Early Marker of Alzheimer's Disease.Brain-to-blood elimination of 24S-hydroxycholesterol from rat brain is mediated by organic anion transporting polypeptide 2 (oatp2) at the blood-brain barrier.
P2860
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P2860
Neurotoxicity of 24-hydroxycholesterol, an important cholesterol elimination product of the brain, may be prevented by vitamin E and estradiol-17beta.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Neurotoxicity of 24-hydroxycho ...... itamin E and estradiol-17beta.
@en
type
label
Neurotoxicity of 24-hydroxycho ...... itamin E and estradiol-17beta.
@en
prefLabel
Neurotoxicity of 24-hydroxycho ...... itamin E and estradiol-17beta.
@en
P2093
P356
P1476
Neurotoxicity of 24-hydroxycho ...... itamin E and estradiol-17beta.
@en
P2093
P2888
P304
P356
10.1007/S007020170068
P577
2001-01-01T00:00:00Z
P6179
1015065332