Interleukin-13 upregulates eotaxin expression in airway epithelial cells by a STAT6-dependent mechanism.
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Coordinate regulation of GATA-3 and Th2 cytokine gene expression by the RNA-binding protein HuRThe adipocyte fatty acid-binding protein aP2 is required in allergic airway inflammationPreclinical development of CAT-354, an IL-13 neutralizing antibody, for the treatment of severe uncontrolled asthmaAnti-IL5 therapy for asthma and beyondRole of infection in the development and exacerbation of asthmaRapid activation of nuclear factor-kappaB in airway epithelium in a murine model of allergic airway inflammationAn alternate STAT6-independent pathway promotes eosinophil influx into blood during allergic airway inflammation.SHP-1 deficiency and increased inflammatory gene expression in PBMCs of multiple sclerosis patients.Expression of IL-4 receptor alpha on smooth muscle cells is not necessary for development of experimental allergic asthma.Leukotriene D4 and interleukin-13 cooperate to increase the release of eotaxin-3 by airway epithelial cells.Mechanisms of eosinophil recruitment and activation.IL-13 regulates cilia loss and foxj1 expression in human airway epithelium.Oleanolic acid controls allergic and inflammatory responses in experimental allergic conjunctivitisPathogenesis of nasal polyposisEosinophil function in allergic inflammation: from bone marrow to tissue response.Elevated H3K18 acetylation in airway epithelial cells of asthmatic subjects.STAT6 expression in T cells, alveolar macrophages and bronchial biopsies of normal and asthmatic subjects.Alternaria fungus induces the production of GM-CSF, interleukin-6 and interleukin-8 and calcium signaling in human airway epithelium through protease-activated receptor 2.IL-13-induced changes in endogenous glucocorticoid metabolism in the lung regulate the proasthmatic response.The proximal STAT6 and NF-kappaB sites are responsible for IL-13- and TNF-alpha-induced RhoA transcriptions in human bronchial smooth muscle cells.Intrinsic defect in T cell production of interleukin (IL)-13 in the absence of both IL-5 and eotaxin precludes the development of eosinophilia and airways hyperreactivity in experimental asthma.Human leukocyte antigen-G expression in differentiated human airway epithelial cells: lack of modulation by Th2-associated cytokines.Genome-Wide Methylation Study Identifies an IL-13-induced Epigenetic Signature in Asthmatic Airways.Epithelial eotaxin-2 and eotaxin-3 expression: relation to asthma severity, luminal eosinophilia and age at onsetWheezing and itching: The requirement for STAT proteins in allergic inflammationCell-penetrating peptides and proteins: new inhibitors of allergic airways disease.Interleukin-4, interleukin-13, signal transducer and activator of transcription factor 6, and allergic asthma.Evaluation of nasal epithelium sampling as a tool in the preclinical development of siRNA-based therapeutics for asthma.Macrophages of multiple sclerosis patients display deficient SHP-1 expression and enhanced inflammatory phenotypeE3 ubiquitin ligase Cbl-b suppresses proallergic T cell development and allergic airway inflammationPI3K and STAT3: a new alliance.The airway epithelium in asthma.Resveratrol and its metabolites modulate cytokine-mediated induction of eotaxin-1 in human pulmonary artery endothelial cells.Airway epithelium interactions with aeroallergens: role of secreted cytokines and chemokines in innate immunity.Cooperative activation of CCL5 expression by TLR3 and tumor necrosis factor-alpha or interferon-gamma through nuclear factor-kappaB or STAT-1 in airway epithelial cells.Hookworm-induced persistent changes to the immunological environment of the lung.IL-13 alters mucociliary differentiation and ciliary beating of human respiratory epithelial cellsMuramyldipeptide modulates CXCL-8 release of BEAS-2B cells via NOD2.Control of eotaxin-1 expression and release by resveratrol and its metabolites in culture human pulmonary artery endothelial cells.Lysophosphatidic acid enhances interleukin-13 gene expression and promoter activity in T cells.
P2860
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P2860
Interleukin-13 upregulates eotaxin expression in airway epithelial cells by a STAT6-dependent mechanism.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Interleukin-13 upregulates eot ...... y a STAT6-dependent mechanism.
@en
type
label
Interleukin-13 upregulates eot ...... y a STAT6-dependent mechanism.
@en
prefLabel
Interleukin-13 upregulates eot ...... y a STAT6-dependent mechanism.
@en
P2093
P2860
P356
P1476
Interleukin-13 upregulates eot ...... by a STAT6-dependent mechanism
@en
P2093
Casolaro V
Kurosawa S
Matsukura S
Schindler U
Schleimer RP
P2860
P304
P356
10.1165/AJRCMB.24.6.4351
P50
P577
2001-06-01T00:00:00Z