Why intracellular parasitism need not be a degrading experience for Mycobacterium.
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Killing of Mycobacterium avium subspecies paratuberculosis within macrophagesMetronidazole therapy in mice infected with tuberculosisMycobacterium tuberculosis catalase and peroxidase activities and resistance to oxidative killing in human monocytes in vitroInnate gene repression associated with Mycobacterium bovis infection in cattle: toward a gene signature of diseaseMycobacterium's arrest of phagosome maturation in macrophages requires Rab5 activity and accessibility to iron.Mycobacterium bovis-infected cervine alveolar macrophages secrete lymphoreactive lipid antigens.Correction of the iron overload defect in beta-2-microglobulin knockout mice by lactoferrin abolishes their increased susceptibility to tuberculosis.PknG supports mycobacterial adaptation in acidic environment.An in vitro model of the leukocyte interactions associated with granuloma formation in Mycobacterium tuberculosis infection.Apoptosis facilitates antigen presentation to T lymphocytes through MHC-I and CD1 in tuberculosis.
P2860
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P2860
Why intracellular parasitism need not be a degrading experience for Mycobacterium.
description
1997 nî lūn-bûn
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1997年の論文
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1997年学术文章
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1997年学术文章
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1997年学术文章
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1997年学术文章
@zh-my
1997年学术文章
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1997年學術文章
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1997年學術文章
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1997年學術文章
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name
Why intracellular parasitism need not be a degrading experience for Mycobacterium.
@en
type
label
Why intracellular parasitism need not be a degrading experience for Mycobacterium.
@en
prefLabel
Why intracellular parasitism need not be a degrading experience for Mycobacterium.
@en
P2093
P2860
P356
P1476
Why intracellular parasitism need not be a degrading experience for Mycobacterium.
@en
P2093
D G Russell
S Sturgill-Koszycki
T Vanheyningen
U E Schaible
P2860
P304
P356
10.1098/RSTB.1997.0114
P407
P577
1997-09-01T00:00:00Z