In situ localization of C3 synthesis in experimental acute renal allograft rejection.
about
Therapeutic regulation of complement in patients with renal disease - where is the promise?The role of complement in the pathogenesis of renal ischemia-reperfusion injury and fibrosisLocal complement C3 expression is upregulated in humoral and cellular rejection of renal allografts.Hemolytic uremic syndrome: a fatal outcome after kidney and liver transplantation performed to correct factor h gene mutation.Deficiency of C4 from donor or recipient mouse fails to prevent renal allograft rejectionLocally produced complement and its role in renal allograft rejection.Complement involvement in kidney diseases: From physiopathology to therapeutical targetingDepression of Complement Regulatory Factors in Rat and Human Renal Grafts Is Associated with the Progress of Acute T-Cell Mediated Rejection.Allograft rejection: effect of local synthesis of complement.New concepts of complement in allorecognition and graft rejectionThe role of complement and Toll-like receptors in organ transplantation.Locally produced C5a binds to T cell-expressed C5aR to enhance effector T-cell expansion by limiting antigen-induced apoptosis.Molecular structure and expression of anthropic, ovine, and murine forms of complement receptor type 2.Renal interstitial cells, proteinuria and progression of lupus nephritis: new frontiers for old factors.The innate immune system and transplantationThe complement cascade and renal disease.The structure, genetic polymorphisms, expression and biological functions of complement receptor type 1 (CR1/CD35).Evolving paradigms that determine the fate of an allograft.siRNA technology in kidney transplantation: current status and future potential.Complement gene expression in human cardiac allograft biopsies as a correlate of histologic grade of injury.Update on ischemia-reperfusion injury in kidney transplantation: Pathogenesis and treatment.The muscle contraction mode determines lymphangiogenesis differentially in rat skeletal and cardiac muscles by modifying local lymphatic extracellular matrix microenvironments.Nontransgenic hyperexpression of a complement regulator in donor kidney modulates transplant ischemia/reperfusion damage, acute rejection, and chronic nephropathy.Which pathways trigger the role of complement in ischaemia/reperfusion injury?Differential allograft gene expression in acute cellular rejection and recurrence of hepatitis C after liver transplantation.Shift of C3 deposition from localization in the glomerulus into the tubulo-interstitial compartment in the absence of secreted IgM in immune complex glomerulonephritis.The membrane attack complex, C5b-9, up regulates collagen gene expression in renal tubular epithelial cells.Complement Alternative Pathway Deficiency in Recipients Protects Kidney Allograft From Ischemia/Reperfusion Injury and Alloreactive T Cell Response.Banff 2003 meeting report: new diagnostic insights and standards.
P2860
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P2860
In situ localization of C3 synthesis in experimental acute renal allograft rejection.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
In situ localization of C3 synthesis in experimental acute renal allograft rejection.
@en
type
label
In situ localization of C3 synthesis in experimental acute renal allograft rejection.
@en
prefLabel
In situ localization of C3 synthesis in experimental acute renal allograft rejection.
@en
P2093
P2860
P1476
In situ localization of C3 synthesis in experimental acute renal allograft rejection.
@en
P2093
P2860
P304
P356
10.1016/S0002-9440(10)64596-8
P407
P577
2000-09-01T00:00:00Z