Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.
about
The mitochondrial permeability transition pore and its role in cell deathThe dual role of calcium as messenger and stressor in cell damage, death, and survival.Direct evidence for tumor necrosis factor-induced mitochondrial reactive oxygen intermediates and their involvement in cytotoxicityBcl-2 and Bcl-X(L) block thapsigargin-induced nitric oxide generation, c-Jun NH(2)-terminal kinase activity, and apoptosisAlpha7 nicotinic receptor activation inhibits ethanol-induced mitochondrial dysfunction, cytochrome c release and neurotoxicity in primary rat hippocampal neuronal cultures.Reactive oxygen/nitrogen species and their functional correlations in neurodegenerative diseases.A2E and blue light in the retina: the paradigm of age-related macular degeneration.Nuclear HMGA1 nonhistone chromatin proteins directly influence mitochondrial transcription, maintenance, and function.Human hsp27, Drosophila hsp27 and human alphaB-crystallin expression-mediated increase in glutathione is essential for the protective activity of these proteins against TNFalpha-induced cell deathAllicin Induces Calcium and Mitochondrial Dysregulation Causing Necrotic Death in Leishmania.Pinusolide and 15-methoxypinusolidic acid attenuate the neurotoxic effect of staurosporine in primary cultures of rat cortical cellsProteolytic-antiproteolytic balance and its regulation in carcinogenesis.Immunohistochemically detectable bcl-2 expression in colorectal carcinoma: correlation with tumour stage and patient survivalRecessive mutations in a common pathway block thymocyte apoptosis induced by multiple signals.Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions.Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death.Reactive oxygen species (ROS)-induced ROS release: a new phenomenon accompanying induction of the mitochondrial permeability transition in cardiac myocytes.Apoptosis or necrosis following Photofrin photosensitization: influence of the incubation protocol.Bcl-2 potentiates the maximal calcium uptake capacity of neural cell mitochondriaHMGA1 levels influence mitochondrial function and mitochondrial DNA repair efficiencySynchronism in mitochondrial ROS flashes, membrane depolarization and calcium sparks in human carcinoma cells.Cytoprotective effect of eckol against oxidative stress-induced mitochondrial dysfunction: involvement of the FoxO3a/AMPK pathway.Activation of CPP32-like protease in tumor necrosis factor-induced apoptosis is dependent on mitochondrial function.Novel path to apoptosis: small transmembrane pores created by staphylococcal alpha-toxin in T lymphocytes evoke internucleosomal DNA degradation.Role of calcium-induced mitochondrial hydroperoxide in induction of apoptosis of RBL2H3 cells with eicosapentaenoic acid treatment.Apoptosis: a modulator of cellular homeostasis and disease states.Control of apoptosis by the cellular ATP level.Apoptosis overview emphasizing the role of oxidative stress, DNA damage and signal-transduction pathways.Reduced capacitative calcium entry correlates with vesicle accumulation and apoptosis.Nitric oxide and its congeners in mitochondria: implications for apoptosis.Effects of radiation on Ca2+ signaling in salivary epithelial cell lines transfected with Bcl-2 and Bcl-XL.Shuttling of calcium between endoplasmic reticulum and mitochondria in the renal vasculature.Light-induced photoreceptor apoptosis in vivo requires neuronal nitric-oxide synthase and guanylate cyclase activity and is caspase-3-independent.Calcium is a key signaling molecule in beta-lapachone-mediated cell death.Insight into age-related macular degeneration: new vision in sight.Early mitochondrial activation and cytochrome c up-regulation during apoptosis.Mitochondrial stress-induced calcium signaling, phenotypic changes and invasive behavior in human lung carcinoma A549 cells.Increased oxidative stress and apoptosis in acute puromycin aminonucleoside nephrosis.Overexpression of Gsalpha compensates for myocyte loss in diabetic cardiomyopathy.Mitochondrial creatine kinase is a prime target of peroxynitrite-induced modification and inactivation.
P2860
Q29614183-8A173CC4-0F2A-4B1D-9332-6DCCB2598C49Q33726729-7D4508E8-A3E2-4CA6-94BE-B2182D90F887Q33948462-7A1C405A-57E7-4D77-BD33-09B20E41AD40Q33958973-EFF8604A-0427-44A6-BE17-06E3B16158F1Q34133440-D51A64E7-E066-40D0-827C-BE5F0BDCAC4CQ34243986-32AF9684-D5EA-4539-BF7C-8DA9AB3DCA6AQ34658953-A002D101-5B11-4E9D-AE87-0767C6723FDEQ35680570-E9FFC8D5-587D-4804-B728-F69F0B578AF0Q35850943-1A89492E-6F9E-46D5-A43B-9768F686ABB3Q35972595-F3D32D7D-6E95-4D97-9F11-1EEC6782B076Q36052485-8BAA5DED-3BBC-4C4F-BC79-CB4E7A549CEFQ36068428-A9AA89A7-3EAE-4B23-BF55-7AF38DB43004Q36081603-026E18B2-2549-4765-B1B2-DB7688935982Q36235008-158D4A7F-DCEE-450B-9EA9-6B2A448E9318Q36255553-C2C2ED34-F66A-435B-B8B9-E4D1BBC6AB1CQ36365164-625BD912-F800-4DBA-B867-B2818B34B1EEQ36368961-19D868A6-D09A-4295-A790-C098EFC9F810Q36817366-AFFB0294-8198-473E-8E90-2AF04BDD0248Q37366855-5B350CCB-48BA-415D-9410-6F6E5B1DD8B0Q37374734-9FC0F66A-4767-4C6C-B4EB-22D426473EB2Q38916727-992500A0-A302-4134-8443-C4B42FDF6398Q39007956-9453782A-1A9F-432F-94DF-17E85F01C999Q39782391-35431306-93A3-4CDC-9912-11ED20945041Q39859690-0A361D5E-E6A1-4A02-BA71-AE4121E64355Q40349374-7F56F679-96AC-44E7-899F-DF07E17E1682Q40858347-7B22840C-5BD1-4A8A-BC08-048554E269B0Q40940073-C2B3A3E0-CBCE-42EC-AD09-17DB301952AEQ40953209-D387F1A9-DA37-43F9-A425-6062E9F5433CQ40967431-26729E7D-127B-406A-8A76-5E3EF16B9BDDQ41886573-9D7B0FAA-7C67-4FA8-B0DD-0BF59AFCEFBFQ42504154-B736936F-BBA3-4544-B4C2-4BBCAD9116B7Q43241606-BE1D7EF8-78A1-4B0C-ABA8-CC0D9B275D5EQ43559246-74481576-56EC-4D27-A19D-5DA5BCF30A08Q43560312-FF02CF5F-D56F-4B51-A55D-9E89A55425F6Q43586539-F46D7716-F9EE-4755-BFAD-C27D68C0FB04Q44198666-117A545B-8ACC-4BE3-ABE5-1E448031AF4AQ44208147-628AC9D5-2EB9-4A7D-A53F-94600997E867Q44868634-C7AE24C0-8C55-4466-854E-0281298DEFD6Q46640265-13A83108-5C59-4961-8483-E4C8E0E1A3EAQ47921911-E32FD391-13F1-465A-8A03-5B7AC259AFAB
P2860
Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.
@en
type
label
Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.
@en
prefLabel
Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.
@en
P2860
P1433
P1476
Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.
@en
P2093
P2860
P304
P356
10.1016/0014-5793(93)81423-W
P407
P577
1993-06-01T00:00:00Z