Evolution of the neurohormonal hypothesis to explain the progression of chronic heart failure.
about
Removal of the cardiac troponin I N-terminal extension improves cardiac function in aged mice.The myocardial matrix and the development and progression of ventricular remodeling.More severe cellular phenotype in human idiopathic dilated cardiomyopathy compared to ischemic heart disease.Novel therapeutic approaches to preserve the right ventricle.The rationale for cardiomyocyte resuscitation in myocardial salvage.Myofilament dysfunction in cardiac disease from mice to men.Acid-base and electrolyte abnormalities in heart failure: pathophysiology and implications.Hypertension up to date: SPRINT to SPYRAL.Role of Angiotensin Receptor-Neprilysin Inhibition in Heart Failure.Central mineralocorticoid receptor blockade improves volume regulation and reduces sympathetic drive in heart failure.Therapeutic targeting of the oncostatin M receptor-β prevents inflammatory heart failure.Brain angiotensin-converting enzyme activity and autonomic regulation in heart failure.Circulating levels of a biomarker of collagen metabolism are associated with health-related quality of life in patients with chronic heart failure.
P2860
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P2860
Evolution of the neurohormonal hypothesis to explain the progression of chronic heart failure.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Evolution of the neurohormonal ...... sion of chronic heart failure.
@en
type
label
Evolution of the neurohormonal ...... sion of chronic heart failure.
@en
prefLabel
Evolution of the neurohormonal ...... sion of chronic heart failure.
@en
P356
P1476
Evolution of the neurohormonal ...... sion of chronic heart failure.
@en
P2093
P356
10.1093/EURHEARTJ/16.SUPPL_F.4
P478
16 Suppl F
P577
1995-07-01T00:00:00Z