Apaf1-deficient cortical neurons exhibit defects in axonal outgrowth.
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The mitochondrial metabolic reprogramming agent trimetazidine as an 'exercise mimetic' in cachectic C26-bearing mice.Modulating the metabolism by trimetazidine enhances myoblast differentiation and promotes myogenesis in cachectic tumor-bearing c26 mice.Acute Noise Exposure Is Associated With Intrinsic Apoptosis in Murine Central Auditory Pathway.Chemotherapeutic Drugs and Mitochondrial Dysfunction: Focus on Doxorubicin, Trastuzumab, and Sunitinib.
P2860
Apaf1-deficient cortical neurons exhibit defects in axonal outgrowth.
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name
Apaf1-deficient cortical neurons exhibit defects in axonal outgrowth.
@en
type
label
Apaf1-deficient cortical neurons exhibit defects in axonal outgrowth.
@en
prefLabel
Apaf1-deficient cortical neurons exhibit defects in axonal outgrowth.
@en
P2093
P2860
P50
P1476
Apaf1-deficient cortical neurons exhibit defects in axonal outgrowth.
@en
P2093
Anna Maria Salvatore
Elisabetta Ferraro
Francesca Molinari
Giuseppe Rosano
Maria Teresa Ciotti
Søs Grønbæk Mathiassen
P2860
P2888
P304
P356
10.1007/S00018-015-1927-X
P577
2015-05-15T00:00:00Z