Complement-mediated injury reversibly disrupts glomerular epithelial cell actin microfilaments and focal adhesions.
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PodocytesHigh glucose and advanced glycosylated end-products affect the expression of alpha-actinin-4 in glomerular epithelial cells.Hepatitis B and Renal Disease.Integrins in kidney development, function, and disease.Experimental membranous nephropathy reduxGlycogen synthase kinase 3β dictates podocyte motility and focal adhesion turnover by modulating paxillin activity: implications for the protective effect of low-dose lithium in podocytopathy.Cytochrome P450 2B1 mediates complement-dependent sublytic injury in a model of membranous nephropathy.Mosaicism of podocyte involvement is related to podocyte injury in females with Fabry diseaseRegulation of adhesive interaction between podocytes and glomerular basement membrane.TRPC channel modulation in podocytes-inching toward novel treatments for glomerular disease.Urinary excretion of viable podocytes in health and renal disease.Transient receptor potential channel 6 (TRPC6) protects podocytes during complement-mediated glomerular disease.The role of complement in membranous nephropathy.A potential role for mechanical forces in the detachment of podocytes and the progression of CKD.Recent advances in the understanding of immune-mediated nephrotic syndrome: diagnostic and prognostic implications.Podocytes … What's Under Yours? (Podocytes and Foot Processes and How They Change in Nephropathy).Mechanical challenges to the glomerular filtration barrier: adaptations and pathway to sclerosis.Terminal complexes of the complement system: new structural insights and their relevance to function.Minimal Change Disease as a Secondary and Reversible Event of a Renal Transplant Case with Systemic Lupus Erythematosus.Astragaloside IV attenuates complement membranous attack complex induced podocyte injury through the MAPK pathway.A comprehensive narrative review of diagnostic biomarkers in human primary membranous nephropathy.Production of a new model of slowly progressive Heymann nephritis.Role of guanine nucleotide exchange factor-H1 in complement-mediated RhoA activation in glomerular epithelial cells.Complement C5b-9 membrane attack complex increases expression of endoplasmic reticulum stress proteins in glomerular epithelial cells.p38 mitogen-activated protein kinase protects glomerular epithelial cells from complement-mediated cell injury.The actin cytoskeleton facilitates complement-mediated activation of cytosolic phospholipase A2.Recent Progress in the Pathogenesis of Nephrotic Proteinuria
P2860
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P2860
Complement-mediated injury reversibly disrupts glomerular epithelial cell actin microfilaments and focal adhesions.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Complement-mediated injury rev ...... filaments and focal adhesions.
@en
type
label
Complement-mediated injury rev ...... filaments and focal adhesions.
@en
prefLabel
Complement-mediated injury rev ...... filaments and focal adhesions.
@en
P2093
P1433
P1476
Complement-mediated injury rev ...... ofilaments and focal adhesions
@en
P2093
P304
P356
10.1046/J.1523-1755.1999.00407.X
P407
P577
1999-05-01T00:00:00Z
P5875
P6179
1033212836