Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon. - Wikidata - wikimedia - TriplyDB

Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

http://www.wikidata.org/entity/Q40974496

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Stromal Hedgehog signalling is downregulated in colon cancer and its restoration restrains tumour growth.Epigenetic regulation of the intestinal epithelium Role of the Wnt/β-catenin pathway in gastric cancer: An in-depth literature review FoxF1 and FoxL1 link hedgehog signaling and the control of epithelial proliferation in the developing stomach and intestine Comparing the DNA hypermethylome with gene mutations in human colorectal cancer.The expanding role of mouse genetics for understanding human biology and disease Functional exploration of the adult ovarian granulosa cell tumor-associated somatic FOXL2 mutation p.Cys134Trp (c.402C>G).The "O" class: crafting clinical care with FoxO transcription factors.Forkhead box L1 is frequently downregulated in gallbladder cancer and inhibits cell growth through apoptosis induction by mitochondrial dysfunction.Foxl1 is a marker of bipotential hepatic progenitor cells in mice.Modifier genes and the plasticity of genetic networks in mice.Epithelial BMP signaling is required for proper specification of epithelial cell lineages and gastric endocrine cells.FGF10 signaling controls stomach morphogenesis.The art and design of genetic screens: mouse.Foxl1-expressing mesenchymal cells constitute the intestinal stem cell niche Wnt signaling and gastrointestinal tumorigenesis in mouse models.The Wnt signaling pathway: aging gracefully as a protectionist?DNA Hypomethylation Contributes to Genomic Instability and Intestinal Cancer Initiation.Clever cancer strategies with FoxO transcription factors.FOXL1, a novel candidate tumor suppressor, inhibits tumor aggressiveness and predicts outcome in human pancreatic cancer.Loss of mesenchymal bone morphogenetic protein signaling leads to development of reactive stroma and initiation of the gastric neoplastic cascade A fork in the path: Developing therapeutic inroads with FoxO proteins.Estrogen receptor α or β loss in the colon of Min/+ mice promotes crypt expansion and impairs TGFβ and HNF3β signaling.Foxl1 inhibits tumor invasion and predicts outcome in human renal cancer APC and its modifiers in colon cancer.SHP-2 phosphatase contributes to KRAS-driven intestinal oncogenesis but prevents colitis-associated cancer development.What are the best routes to effectively model human colorectal cancer?Somatic recombination in adult tissues: What is there to learn?Low level of FOXL1 indicates a worse prognosis for gastric cancer patients.Giving APCmin tumours a SPARC.The Emerging Roles of Forkhead Box (FOX) Proteins in Osteosarcoma.Screening Driving Transcription Factors in the Processing of Gastric Cancer.Foxl1-deficient mice exhibit aberrant epithelial cell positioning resulting from dysregulated EphB/EphrinB expression in the small intestine

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Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

http://www.wikidata.org/entity/Q40974496

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

@zh-tw

2005年论文

@wuu

2005年论文

@zh

2005年论文

@zh-cn

name

Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

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type

label

Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

@en

prefLabel

Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

@en

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Genes & Development

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Foxl1 is a mesenchymal Modifier of Min in carcinogenesis of stomach and colon.

@en

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Emma E Furth

http://www.w3.org/2001/XMLSchema#string

Jonathan P Katz

http://www.w3.org/2001/XMLSchema#string

Klaus H Kaestner

http://www.w3.org/2001/XMLSchema#string

Nathalie Perreault

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Sara D Sackett

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P2860

Clustered arrangement of winged helix genes fkh-6 and MFH-1: possible implications for mesoderm development

Loss of the PLA2G2A gene in a sporadic colorectal tumor of a patient with a PLA2G2A germline mutation and absence of PLA2G2A germline alterations in patients with FAP

beta-catenin signaling and cancer

The mesenchymal winged helix transcription factor Fkh6 is required for the control of gastrointestinal proliferation and differentiation

Foxl1 controls the Wnt/beta-catenin pathway by modulating the expression of proteoglycans in the gut

The Min (multiple intestinal neoplasia) mutation: its effect on gut epithelial cell differentiation and interaction with a modifier system

Mom1 is a semi-dominant modifier of intestinal adenoma size and multiplicity in Min/+ mice

Secretory phospholipase Pla2g2a confers resistance to intestinal tumorigenesis

A dominant mutation that predisposes to multiple intestinal neoplasia in the mouse

A modifying locus for familial adenomatous polyposis may be present on chromosome 1p35-p36

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311-315

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scholarly article

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10.1101/GAD.1260605

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3

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19

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2005-01-13T00:00:00Z

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15650110

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546508

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