Activation of aryl hydrocarbon receptor dissociates fatty liver from insulin resistance by inducing fibroblast growth factor 21.
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Long-term in vivo polychlorinated biphenyl 126 exposure induces oxidative stress and alters proteomic profile on islets of Langerhans.A cellular model to study drug-induced liver injury in nonalcoholic fatty liver disease: Application to acetaminophenHost-microbiome interactions: the aryl hydrocarbon receptor and the central nervous systemFGF21 protects against ox-LDL induced apoptosis through suppressing CHOP expression in THP1 macrophage derived foam cells.Inhibition of miR-29 has a significant lipid-lowering benefit through suppression of lipogenic programs in liver.Exchange of polar lipids from adults to neonates in Daphnia magna: Perturbations in sphingomyelin allocation by dietary lipids and environmental toxicants.Perturbations in polar lipids, starvation survival and reproduction following exposure to unsaturated fatty acids or environmental toxicants in Daphnia magna.Aryl Hydrocarbon Receptor Plays Protective Roles against High Fat Diet (HFD)-induced Hepatic Steatosis and the Subsequent Lipotoxicity via Direct Transcriptional Regulation of Socs3 Gene ExpressionHepatic Aryl Hydrocarbon Receptor Attenuates Fibroblast Growth Factor 21 ExpressionMechanisms of xenobiotic receptor activation: Direct vs. indirect.A Fibroblast Growth Factor 21-Pregnane X Receptor Pathway Downregulates Hepatic CYP3A4 in Nonalcoholic Fatty Liver Disease.Emerging roles of xenobiotic detoxification enzymes in metabolic diseases.Hepatic Overexpression of CD36 Improves Glycogen Homeostasis and Attenuates High-Fat Diet-Induced Hepatic Steatosis and Insulin Resistance.Liver PPARα is crucial for whole-body fatty acid homeostasis and is protective against NAFLD.Dose-Dependent Metabolic Reprogramming and Differential Gene Expression in TCDD-Elicited Hepatic Fibrosis.AhR and SHP regulate phosphatidylcholine and S-adenosylmethionine levels in the one-carbon cycle.A compromised liver alters polychlorinated biphenyl-mediated toxicity.The Biological Clock: A Pivotal Hub in Non-alcoholic Fatty Liver Disease Pathogenesis.Aryl hydrocarbon receptor-fibroblast growth factor 21 dissociation of fatty liver from insulin resistance: A timely matter?Fibroblast growth factor 21 inhibits atherosclerosis in apoE-/- mice by ameliorating Fas-mediated apoptosis
P2860
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P2860
Activation of aryl hydrocarbon receptor dissociates fatty liver from insulin resistance by inducing fibroblast growth factor 21.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Activation of aryl hydrocarbon ...... g fibroblast growth factor 21.
@en
type
label
Activation of aryl hydrocarbon ...... g fibroblast growth factor 21.
@en
prefLabel
Activation of aryl hydrocarbon ...... g fibroblast growth factor 21.
@en
P2093
P2860
P356
P1433
P1476
Activation of aryl hydrocarbon ...... g fibroblast growth factor 21.
@en
P2093
Pengcheng Wang
Wojciech G Garbacz
Xiaochao Ma
P2860
P304
P356
10.1002/HEP.27719
P407
P577
2015-02-27T00:00:00Z