MHC class II gene silencing in trophoblast cells is caused by inhibition of CIITA expression. - Wikidata - wikimedia - TriplyDB

MHC class II gene silencing in trophoblast cells is caused by inhibition of CIITA expression.

MHC class II gene silencing in trophoblast cells is caused by inhibition of CIITA expression.

http://www.wikidata.org/entity/Q40979361

about

Associations and interactions between bare lymphocyte syndrome factors Class II transactivator: mastering the art of major histocompatibility complex expression Regulation of major histocompatibility complex class II gene expression in trophoblast cells Interferon gamma in successful pregnancies.Gamma interferon fails to induce expression of indoleamine 2,3-dioxygenase and does not control the growth of Chlamydophila abortus in BeWo trophoblast cells.Histone deacetylases inhibit IFN-gamma-inducible gene expression in mouse trophoblast cells Fetomaternal immunotolerance.Histone deacetylase inhibitors activate CIITA and MHC class II antigen expression in diffuse large B-cell lymphoma.Kinetics of a gamma interferon response: expression and assembly of CIITA promoter IV and inhibition by methylation.Human leucocyte antigen (HLA) expression of primary trophoblast cells and placental cell lines, determined using single antigen beads to characterize allotype specificities of anti-HLA antibodies.Class II transactivator (CIITA) deficiency in tumor cells: complicated mechanisms or not?Genomewide association study of HLA alloimmunization in previously pregnant blood donors.

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MHC class II gene silencing in trophoblast cells is caused by inhibition of CIITA expression.

http://www.wikidata.org/entity/Q40979361

description

1998 nî lūn-bûn

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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1998年论文

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1998年论文

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American Journal of Reproductive Immunology

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A C Morris

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J L Riley

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J M Boss

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W H Fleming

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P2860

RFXAP, a novel subunit of the RFX DNA binding complex is mutated in MHC class II deficiency

Complementation cloning of an MHC class II transactivator mutated in hereditary MHC class II deficiency (or bare lymphocyte syndrome)

Activation of class II MHC genes requires both the X box region and the class II transactivator (CIITA)

The DNA-binding defect observed in major histocompatibility complex class II regulatory mutants concerns only one member of a family of complexes binding to the X boxes of class II promoters

Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

A novel multigene family may encode odorant receptors: a molecular basis for odor recognition

A novel DNA-binding regulatory factor is mutated in primary MHC class II deficiency (bare lymphocyte syndrome)

Stat recruitment by tyrosine-phosphorylated cytokine receptors: an ordered reversible affinity-driven process

Targeted disruption of the Stat1 gene in mice reveals unexpected physiologic specificity in the JAK-STAT signaling pathway

Conserved upstream sequences of human class II major histocompatibility genes enhance expression of class II genes in wild-type but not mutant B-cell lines.

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385-394

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40

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