Multiple hemopoietic defects and lymphoid hyperplasia in mice lacking the transcriptional activation domain of the c-Rel protein.
about
Rel/NF-kappaB can trigger the Notch signaling pathway by inducing the expression of Jagged1, a ligand for Notch receptorsUnexpected functions of nuclear factor-κB during germinal center B-cell development: implications for lymphomagenesisThe c-Rel transcription factor and B-cell proliferation: a deal with the devilCytosolic glucocorticoid receptor interaction with nuclear factor-kappa B proteins in rat liver cellsHistone acetyltransferase p300 is a coactivator for transcription factor REL and is C-terminally truncated in the human diffuse large B-cell lymphoma cell line RC-K8.Genetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts.Germinal center B cell maintenance and differentiation are controlled by distinct NF-κB transcription factor subunits.The clinical implication and molecular mechanism of preferential IL-4 production by modified glycolipid-stimulated NKT cells.NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions.Critical role of cRel subunit of NF-κB in sepsis survival.NF-κB in immunobiology.Loss of c-REL but not NF-κB2 prevents autoimmune disease driven by FasL mutation.The c-Rel Transcription Factor in Development and Disease.Signaling pathways in Th2 development.Functional redundancy of the nuclear factor kappa B inhibitors I kappa B alpha and I kappa B betaAssociation of REL polymorphisms and outcome of patients with septic shock.Dendritic cells for active anti-cancer immunotherapy: targeting activation pathways through genetic modification.Differential requirements for the canonical NF-κB transcription factors c-REL and RELA during the generation and activation of mature B cells.Lymphangiogenesis, myeloid cells and inflammation.A single NFκB system for both canonical and non-canonical signaling.Regulation of NF-κB by TNF family cytokines.Differing Requirements for MALT1 Function in Peripheral B Cell Survival and Differentiation.Genomic organization and expression of the rearranged REL proto-oncogene in the human B-cell lymphoma cell line RC-K8.Celebrating 25 years of NF-κB research.Loss of p16Ink4a with retention of p19Arf predisposes mice to tumorigenesis.An optimal range of transcription potency is necessary for efficient cell transformation by c-Rel to ensure optimal nuclear localization and gene-specific activation.NF-κB2/p100 deficiency impairs immune responses to T-cell-independent type 2 antigens.Combined loss of cRel/p50 subunits of NF-κB leads to impaired innate host response in sepsis
P2860
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P2860
Multiple hemopoietic defects and lymphoid hyperplasia in mice lacking the transcriptional activation domain of the c-Rel protein.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Multiple hemopoietic defects a ...... n domain of the c-Rel protein.
@en
type
label
Multiple hemopoietic defects a ...... n domain of the c-Rel protein.
@en
prefLabel
Multiple hemopoietic defects a ...... n domain of the c-Rel protein.
@en
P2093
P2860
P356
P1476
Multiple hemopoietic defects a ...... n domain of the c-Rel protein.
@en
P2093
D Carrasco
P2860
P304
P356
10.1084/JEM.187.7.973
P407
P577
1998-04-01T00:00:00Z