WEE1 inhibition in pancreatic cancer cells is dependent on DNA repair status in a context dependent manner.
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Pharmacotherapeutic Management of Pancreatic Ductal Adenocarcinoma: Current and Emerging Concepts.MEK inhibitors cobimetinib and trametinib, regressed a gemcitabine-resistant pancreatic-cancer patient-derived orthotopic xenograft (PDOX)Mutational status of TP53 defines the efficacy of Wee1 inhibitor AZD1775 in KRAS-mutant non-small cell lung cancer.Elevated HuR in Pancreas Promotes a Pancreatitis-like Inflammatory Microenvironment that Facilitates Tumor Development.Directing the use of DDR kinase inhibitors in cancer treatment.CRISPR Knockout of the HuR Gene Causes a Xenograft Lethal Phenotype.Augmentation of the therapeutic efficacy of WEE1 kinase inhibitor AZD1775 by inhibiting the YAP-E2F1-DNA damage response pathway axis.Augmented antitumor activity by olaparib plus AZD1775 in gastric cancer through disrupting DNA damage repair pathways and DNA damage checkpoint.
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P2860
WEE1 inhibition in pancreatic cancer cells is dependent on DNA repair status in a context dependent manner.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
@zh-my
2016年学术文章
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2016年學術文章
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2016年學術文章
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2016年學術文章
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name
WEE1 inhibition in pancreatic ...... in a context dependent manner.
@en
type
label
WEE1 inhibition in pancreatic ...... in a context dependent manner.
@en
prefLabel
WEE1 inhibition in pancreatic ...... in a context dependent manner.
@en
P2093
P2860
P356
P1433
P1476
WEE1 inhibition in pancreatic ...... in a context dependent manner.
@en
P2093
Charles J Yeo
Emanuela Dylgjeri
Jonathan R Brody
Jordan M Winter
Michael J Pishvaian
Nicole C Mambelli-Lisboa
Saswati N Chand
P2860
P2888
P356
10.1038/SREP33323
P407
P577
2016-09-12T00:00:00Z