Increased levels of substance P in the myenteric plexus of Trichinella-infected rats.
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An Update on Post-infectious Irritable Bowel Syndrome: Role of Genetics, Immune Activation, Serotonin and Altered MicrobiomeDiverticulosis today: unfashionable and still under-researchedGastrointestinal Parasites and the Neural Control of Gut FunctionsIntestinal inflammation and activation of sensory nerve pathways: a functional and morphological study in the nematode infected ratColitis affects the smooth muscle and neural response to motilin in the rabbit antrumRole of nerves in enteric infection.Neurokinin-1 receptor expression in inflammatory bowel disease: molecular quantitation and localisation.Increased rectal mucosal enteroendocrine cells, T lymphocytes, and increased gut permeability following acute Campylobacter enteritis and in post-dysenteric irritable bowel syndromeNeutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance PTachykinin inhibition of acid-induced gastric hyperaemia in the rat.Increased substance P responses in dorsal root ganglia and intestinal macrophages during Clostridium difficile toxin A enteritis in rats.Role of substance P and the neurokinin 1 receptor in acute pancreatitis and pancreatitis-associated lung injury.The Therapeutic Potential of Targeting Substance P/NK-1R Interactions in Inflammatory CNS DisordersCP-96,345, a substance P antagonist, inhibits rat intestinal responses to Clostridium difficile toxin A but not cholera toxin.Alterations in intestinal contractility during inflammation are caused by both smooth muscle damage and specific receptor-mediated mechanisms.Neutral endopeptidase (EC 3.4.24.11) downregulates the onset of intestinal inflammation in the nematode infected mouse.Delayed-type hypersensitivity-induced increase in vascular permeability in the mouse small intestine: inhibition by depletion of sensory neuropeptides and NK1 receptor blockade.The Gastric Ganglion of Octopus vulgaris: Preliminary Characterization of Gene- and Putative Neurochemical-Complexity, and the Effect of Aggregata octopiana Digestive Tract Infection on Gene Expression.Colonic levels of vasoactive intestinal peptide decrease during infection and exogenous VIP protects epithelial mitochondria against the negative effects of IFNγ and TNFα induced during Citrobacter rodentium infection
P2860
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P2860
Increased levels of substance P in the myenteric plexus of Trichinella-infected rats.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Increased levels of substance P in the myenteric plexus of Trichinella-infected rats.
@en
type
label
Increased levels of substance P in the myenteric plexus of Trichinella-infected rats.
@en
prefLabel
Increased levels of substance P in the myenteric plexus of Trichinella-infected rats.
@en
P2093
P1433
P1476
Increased levels of substance P in the myenteric plexus of Trichinella-infected rats.
@en
P2093
Blennerhassett P
Collins SM
P304
P407
P577
1992-06-01T00:00:00Z