A localized absence of interleukin-4 triggers periodontal disease activity: a novel hypothesis.
about
A genome-wide association study identifies GLT6D1 as a susceptibility locus for periodontitisInterleukin-4 inhibition of interleukin-1-induced expression of matrix metalloproteinase-3 (MMP-3) is independent of lipoxygenase and PPARgamma activation in human gingival fibroblasts.The expressions of inflammatory factors and tissue inhibitor of matrix metalloproteinase-2 in human chronic periodontitis with type 2 diabetes mellitus.Serum immunoglobulin G (IgG) and IgG subclass responses to the RgpA-Kgp proteinase-adhesin complex of Porphyromonas gingivalis in adult periodontitisDestructive periodontitis lesions are determined by the nature of the lymphocytic response.Association of susceptible genotypes to periodontal disease with the clinical outcome and tooth survival after non-surgical periodontal therapy: A systematic review and meta-analysis.Association between Polymorphisms in Interleukins 4 and 13 Genes and Chronic Periodontitis in a Han Chinese PopulationAlterations in phagocyte function and periodontal infection.T-cell phenotype as a risk factor for periodontal disease.Association between interleukin-4 gene -590 c/t, -33 c/t, and 70-base-pair polymorphisms and periodontitis susceptibility: a meta-analysis.Interleukin-4 suppresses IL-1-induced expression of matrix metalloproteinase-3 in human gingival fibroblasts.T- and B-cell subsets in periodontitis.Interleukin-4 suppression of interleukin-1-induced transcription of collagenase (MMP-1) and stromelysin 1 (MMP-3) in human synovial fibroblasts.Absolute quantification of Aggregatibacter actinomycetemcomitans in patients carrying haplotypes associated with susceptibility to chronic periodontitis: multifaceted evaluation with periodontitis covariants.IL-4 inhibition of IL-1 induced Matrix metalloproteinase-3 (MMP-3) expression in human fibroblasts involves decreased AP-1 activation via negative crosstalk involving of Jun N-terminal kinase (JNK).The role of the host response in periodontal disease progression: implications for future treatment strategies.The acute inflammatory response and the role of phagocytic cells in periodontal health and disease.Interferon-γ, interleukins-6 and -4, and factor XIII-A as indirect markers of the classical and alternative macrophage activation pathways in chronic periodontitis.Periodontopathogens levels and clinical response to periodontal therapy in individuals with the interleukin-4 haplotype associated with susceptibility to chronic periodontitis.Effect of smoking on gingival crevicular fluid cytokine profile during experimental gingivitis.Inflammatory mediators of the terminal dentition in adult and early onset periodontitis.Interleukin-4 gene polymorphisms in Japanese and Caucasian patients with aggressive periodontitis.Genetic polymorphisms and periodontal disease in populations of African descent: A review.Resveratrol Inhibits Periodontitis-Related Bone Loss in Rats Subjected to Cigarette Smoke Inhalation.Polymorphisms and haplotypes in the interleukin-4 gene are associated with chronic periodontitis in a Brazilian population.Possible involvement of IL-21 and IL-10 on salivary IgA levels in chronic periodontitis subjects.Interleukin-4 (C-590T) and interferon-gamma (G5644A) gene polymorphisms in patients with periodontitis.
P2860
Q28943481-A3875108-4DF2-43B3-ADC4-087147B04E32Q30830094-3D13C92A-BBE2-4B2A-8EB7-61F8366F8AC6Q33861361-09811C99-65F2-4C10-8811-34A488F67281Q34004291-A45379BB-E01B-488D-925E-A5461EDC21AEQ34720552-1640F3A9-1D8A-4D7C-8C5F-F35C28A8EAB8Q35848745-78875757-3D5A-406C-A9AF-625139601401Q36022021-CD737F9D-0AC6-4BD7-B7B8-75692BD19BDEQ36831541-12392C51-FA93-4F3D-8677-BE1F86842A75Q36938521-5447A3E4-F263-4BE8-A3F6-8B72C997DAB9Q38230110-ACE0BA9F-A681-4C40-8275-24C2C3AA72FDQ38504663-F49DB907-7689-4FBC-9C69-857A648E88C4Q38562438-30FFAE77-BCA9-43CF-A504-E3CDE36AD229Q38583149-5BEA1533-4FA6-4389-809F-35B42307064CQ38599652-346DD01C-7CA8-4629-B77B-5F2C423D033BQ39162312-754B333C-8810-430C-8560-990460183247Q40864530-C551BA3F-8572-4B46-A43D-CF67D8DEF916Q41754960-95676FC5-D9CD-4811-A2E4-FD82298AFE52Q45207763-E78C4EF2-90DB-426F-A0FB-3DACF4255C5BQ46817160-CACC704D-D6A9-4D64-82DC-4A684D52A557Q47221132-FCAED48C-5277-4488-AAB2-3859A470FA0FQ47743621-18D6A893-C268-4BAC-9F11-CD2DDD99E60BQ47758049-14B093F3-4FA6-45CB-9FAF-50DFF38AAA5DQ48361240-F980B090-33BC-4C2A-9714-A1FFF5B937B0Q50656841-E6DC238D-35C8-4383-8F54-7BA380018C63Q51717487-5E3E911C-D04C-443B-BE01-E54D587A7C12Q54355713-BF723FD8-4DC8-409A-8B29-3DDDFDE2F7A8Q54549068-584347DC-CBAF-46AA-99DA-5C90F9140F46
P2860
A localized absence of interleukin-4 triggers periodontal disease activity: a novel hypothesis.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
A localized absence of interle ...... activity: a novel hypothesis.
@en
type
label
A localized absence of interle ...... activity: a novel hypothesis.
@en
prefLabel
A localized absence of interle ...... activity: a novel hypothesis.
@en
P2093
P1433
P1476
A localized absence of interle ...... activity: a novel hypothesis.
@en
P2093
P304
P356
10.1016/0306-9877(92)90056-I
P577
1992-12-01T00:00:00Z