Deletion of Apoptosis Inhibitor of Macrophage (AIM)/CD5L Attenuates the Inflammatory Response and Infarct Size in Acute Myocardial Infarction.
about
Apoptosis inhibitor of macrophage depletion decreased M1 macrophage accumulation and the incidence of cardiac rupture after myocardial infarction in mice.Monocyte-derived alveolar macrophages drive lung fibrosis and persist in the lung over the life span.Liraglutide repairs the infarcted heart: The role of the SIRT1/Parkin/mitophagy pathway.Endothelial stem cells attenuate cardiac apoptosis via downregulating cardiac microRNA-146a in a rat model of coronary heart disease
P2860
Deletion of Apoptosis Inhibitor of Macrophage (AIM)/CD5L Attenuates the Inflammatory Response and Infarct Size in Acute Myocardial Infarction.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Deletion of Apoptosis Inhibito ...... n Acute Myocardial Infarction.
@en
type
label
Deletion of Apoptosis Inhibito ...... n Acute Myocardial Infarction.
@en
prefLabel
Deletion of Apoptosis Inhibito ...... n Acute Myocardial Infarction.
@en
P2093
P2860
P356
P1476
Deletion of Apoptosis Inhibito ...... n Acute Myocardial Infarction.
@en
P2093
Aya Shiraki
Hiroshi Komoda
Jun-ichi Oyama
Koichi Node
Toshiyuki Nishikido
P2860
P304
P356
10.1161/JAHA.115.002863
P577
2016-04-04T00:00:00Z