Gap junctions promote the bystander effect of herpes simplex virus thymidine kinase in vivo.
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Dexamethasone inhibits the HSV-tk/ ganciclovir bystander effect in malignant glioma cells.Stem cells as cellular vehicles for gene therapy against glioblastomaSafety and biodistribution studies of an HSV multigene vector following intracranial delivery to non-human primates.Therapeutic efficacy of human hepatocyte transplantation in a SCID/uPA mouse model with inducible liver diseaseSuicidal gene therapy in an NF-κB-controlled tumor environment as monitored by a secreted blood reporterThe engineered thymidylate kinase (TMPK)/AZT enzyme-prodrug axis offers efficient bystander cell killing for suicide gene therapy of cancerCharacterization of herpes simplex virus type 1 thymidine kinase mutants engineered for improved ganciclovir or acyclovir activity.Advancement and prospects of tumor gene therapyFusion enzymes containing HSV-1 thymidine kinase mutants and guanylate kinase enhance prodrug sensitivity in vitro and in vivo.The role of gap junction channels during physiologic and pathologic conditions of the human central nervous systemGene therapy and targeted toxins for glioma.Tunneling nanotubes: an alternate route for propagation of the bystander effect following oncolytic viral infection.Gap junctions and cancer: communicating for 50 years.Chapter seven--Cancer treatment with gene therapy and radiation therapy.Influence of drugs on gap junctions in glioma cell lines and primary astrocytes in vitro.Exceptionally potent anti-tumor bystander activity of an scFv:sTRAIL fusion protein with specificity for EGP2 toward target antigen-negative tumor cells.Bovine herpesvirus tegument protein VP22 enhances thymidine kinase/ganciclovir suicide gene therapy for neuroblastomas compared to herpes simplex virus VP22.Mind the Gaps in Tumor Immunity: Impact of Connexin-Mediated Intercellular Connections.NG2 glial cells regulate neuroimmunological responses to maintain neuronal function and survival.Disruption of gap junctions attenuates aminoglycoside-elicited renal tubular cell injury.Suicide gene therapy for chemically induced rat bladder tumor entailing instillation of adenoviral vectors.Influence of vascular endothelial growth factor and radiation on gap junctional intercellular communication in glioblastoma multiforme cell lines.The Novel Roles of Connexin Channels and Tunneling Nanotubes in Cancer Pathogenesis.Gene therapy and targeted toxins for glioma
P2860
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P2860
Gap junctions promote the bystander effect of herpes simplex virus thymidine kinase in vivo.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Gap junctions promote the byst ...... irus thymidine kinase in vivo.
@en
type
label
Gap junctions promote the byst ...... irus thymidine kinase in vivo.
@en
prefLabel
Gap junctions promote the byst ...... irus thymidine kinase in vivo.
@en
P2093
P1433
P1476
Gap junctions promote the byst ...... virus thymidine kinase in vivo
@en
P2093
Björkstrand B
Christensson B
P304
P407
P577
1997-04-01T00:00:00Z