about
The origins of concentric demyelination: self-organization in the human brainIncreased expression of ICAM-1, VCAM-1, MCP-1, and MIP-1 alpha by spinal perivascular macrophages during experimental allergic encephalomyelitis in rats.Expression of inducible nitric oxide synthase and nitrotyrosine in multiple sclerosis lesionsA new formulation of cannabidiol in cream shows therapeutic effects in a mouse model of experimental autoimmune encephalomyelitis.Molecular characterization of antibody specificities against myelin/oligodendrocyte glycoprotein in autoimmune demyelinationEffects of 15-deoxy-delta12,14-prostaglandin J2 (15d-PGJ2) and rosiglitazone on human gammadelta2 T cells.Immune response-mediated protection of adult but not neonatal mice from neuron-restricted measles virus infection and central nervous system disease.Fibrin depletion decreases inflammation and delays the onset of demyelination in a tumor necrosis factor transgenic mouse model for multiple sclerosis.Interleukin-17 mRNA expression in blood and CSF mononuclear cells is augmented in multiple sclerosis.Chemokine receptor antagonism as a new therapy for multiple sclerosis.Myelin basic protein induces neuron-specific toxicity by directly damaging the neuronal plasma membrane.Microparticles in multiple sclerosis and clinically isolated syndrome: effect on endothelial barrier functionCytokines in multiple sclerosis: methodological aspects and pathogenic implications.Oligodendrocyte apoptosis and primary demyelination induced by local TNF/p55TNF receptor signaling in the central nervous system of transgenic mice: models for multiple sclerosis with primary oligodendrogliopathy.Temporal expression and cellular origin of CC chemokine receptors CCR1, CCR2 and CCR5 in the central nervous system: insight into mechanisms of MOG-induced EAE.Redox regulation of cytokine-mediated inhibition of myelin gene expression in human primary oligodendrocytes.Myelin basic protein-primed T cells of female but not male mice induce nitric-oxide synthase and proinflammatory cytokines in microglia: implications for gender bias in multiple sclerosis.Role of very-late antigen-4 (VLA-4) in myelin basic protein-primed T cell contact-induced expression of proinflammatory cytokines in microglial cellsClonal expansions of CD8(+) T cells dominate the T cell infiltrate in active multiple sclerosis lesions as shown by micromanipulation and single cell polymerase chain reactionThe cooling effect on proinflammatory cytokines interferon-gamma, tumor necrosis factor-alpha, and nitric oxide in patients with multiple sclerosis.CB2 cannabinoid receptors as an emerging target for demyelinating diseases: from neuroimmune interactions to cell replacement strategies.Possible involvement of TLRs and hemichannels in stress-induced CNS dysfunction via mastocytes, and glia activation.Molecular tracking of antigen-specific T cell clones in neurological immune-mediated disordersModulation of Rho-Rock signaling pathway protects oligodendrocytes against cytokine toxicity via PPAR-α-dependent mechanismThe role of IL-15 in central nervous system disorders.Role of chemokine CCL2 and its receptor CCR2 in neurodegenerative diseases.Multiple sclerosis: re-expression of a developmental pathway that restricts oligodendrocyte maturation.Purinergic Receptor Expression and Potential Association with Human Embryonic Stem Cell-Derived Oligodendrocyte Progenitor Cell Development.The importance of studying sex differences in disease: The example of multiple sclerosis.Structural dynamics of oligodendrocyte lysis by perforin in culture: relevance to multiple sclerosis.Oligoclonal expansion of memory CD8+ T cells in cerebrospinal fluid from multiple sclerosis patients.Effects of IFN-β1a and IFN-β1b treatment on the expression of cytokines, inducible NOS (NOS type II), and myelin proteins in animal model of multiple sclerosis.Cytokine-directed therapies in multiple sclerosis and experimental autoimmune encephalomyelitis.Circulating interleukin-15 and RANTES chemokine in MS patients: effect of treatment with methylprednisolone in patients with relapse.Myelin-induced microglial neurotoxicity can be controlled by microglial metabotropic glutamate receptors.Remyelination in experimentally demyelinated connexin 32 knockout mice.Frequency, heterogeneity and encephalitogenicity of T cells specific for myelin oligodendrocyte glycoprotein in naive outbred primates.Macrophage and microglial responses to cytokines in vitro: phagocytic activity, proteolytic enzyme release, and free radical production.Identification of autoantibodies associated with myelin damage in multiple sclerosis.Late complications of immune deviation therapy in a nonhuman primate.
P2860
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P2860
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Mechanisms of immune injury in multiple sclerosis.
@en
type
label
Mechanisms of immune injury in multiple sclerosis.
@en
prefLabel
Mechanisms of immune injury in multiple sclerosis.
@en
P2860
P1433
P1476
Mechanisms of immune injury in multiple sclerosis.
@en
P2093
P2860
P304
P356
10.1111/J.1750-3639.1996.TB00853.X
P577
1996-07-01T00:00:00Z