ORE, a eukaryotic minimal essential osmotic response element. The aldose reductase gene in hyperosmotic stress.
about
Tonicity-responsive enhancer binding protein, a rel-like protein that stimulates transcription in response to hypertonicityTransgenic rats reveal functional conservation of regulatory controls between the Fugu isotocin and rat oxytocin genesNeuropathy target esterase catalyzes osmoprotective renal synthesis of glycerophosphocholine in response to high NaClUp-regulation of hypertonicity-activated myo-inositol transporter SMIT1 by the cell volume-sensitive protein kinase SGK1c-Abl mediates high NaCl-induced phosphorylation and activation of the transcription factor TonEBP/OREBPIn vivo regulation of MAP kinases in Ratus norvegicus renal papilla by water loading and restrictionStimulation of GLUT-1 glucose transporter expression in response to hyperosmolarity.Structure and characterization of the mouse UT-A gene (Slc14a2).Signalling and gene regulation by urea and NaCl in the renal medulla.What sets the TonE during osmotic stress?Polyol pathway and diabetic nephropathy revisited: Early tubular cell changes and glomerulopathy in diabetic mice overexpressing human aldose reductaseOsmoregulation in the mammalian kidney: the role of organic osmolytes.Aldose reductase-deficient mice develop nephrogenic diabetes insipidus.NFAT5 is activated by hypoxia: role in ischemia and reperfusion in the rat kidney.Survival in hostile environments: strategies of renal medullary cells.High NaCl-induced activation of CDK5 increases phosphorylation of the osmoprotective transcription factor TonEBP/OREBP at threonine 135, which contributes to its rapid nuclear localization.Dehydration activates an NF-kappaB-driven, COX2-dependent survival mechanism in renal medullary interstitial cells.MAP kinases and the adaptive response to hypertonicity: functional preservation from yeast to mammals.Mutations that reduce its specific DNA binding inhibit high NaCl-induced nuclear localization of the osmoprotective transcription factor NFAT5TAZ suppresses NFAT5 activity through tyrosine phosphorylation.Volume-sensitive K(+)/Cl(-) cotransport in rabbit erythrocytes. Analysis of the rate-limiting activation and inactivation events.Osmotic regulation of a novel flavin-containing monooxygenase in primary cultured cells from rainbow trout (Oncorhynchus mykiss).NFAT5 in cellular adaptation to hypertonic stress - regulations and functional significanceGlucose-specific regulation of aldose reductase in capan-1 human pancreatic duct cells In vitro.The betaine/GABA transporter and betaine: roles in brain, kidney, and liver.Osmolality/salinity-responsive enhancers (OSREs) control induction of osmoprotective genes in euryhaline fish.Osmotic response element enhancer activity. Regulation through p38 kinase and mitogen-activated extracellular signal-regulated kinase kinase.Hypertonic induction of the cell adhesion molecule beta 1-integrin in MDCK cells.Activation of osmolyte pathways in inflammatory myopathy and Duchenne muscular dystrophy points to osmoregulation as a contributing pathogenic mechanism.Osmotic diuretics induce adenosine A1 receptor expression and protect renal proximal tubular epithelial cells against cisplatin-mediated apoptosis.Hypertonicity-induced aquaporin-1 (AQP1) expression is mediated by the activation of MAPK pathways and hypertonicity-responsive element in the AQP1 gene.Hypertonicity-induced expression of aquaporin 3 in MDCK cells.The MKK(3/6)-p38-signaling cascade alters the subcellular distribution of hnRNP A1 and modulates alternative splicing regulation.Ras signaling in the inner medullary cell response to urea and NaCl.Characterization of cis-elements required for osmotic response of rat Na(+)/H(+) exchanger-2 (NHE-2) gene.Osmotic response element is required for the induction of aldose reductase by tumor necrosis factor-alpha.Dominant-negative c-Jun NH2-terminal kinase 2 sensitizes renal inner medullary collecting duct cells to hypertonicity-induced lethality independent of organic osmolyte transport.Identification of a novel cis-element required for the constitutive activity and osmotic response of the rat aldose reductase promoter.Distinct regulation of osmoprotective genes in yeast and mammals. Aldose reductase osmotic response element is induced independent of p38 and stress-activated protein kinase/Jun N-terminal kinase in rabbit kidney cells.Differential control of murine aldose reductase and fibroblast growth factor (FGF)-regulated-1 gene expression in NIH 3T3 cells by FGF-1 treatment and hyperosmotic stress
P2860
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P2860
ORE, a eukaryotic minimal essential osmotic response element. The aldose reductase gene in hyperosmotic stress.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
ORE, a eukaryotic minimal esse ...... e gene in hyperosmotic stress.
@en
type
label
ORE, a eukaryotic minimal esse ...... e gene in hyperosmotic stress.
@en
prefLabel
ORE, a eukaryotic minimal esse ...... e gene in hyperosmotic stress.
@en
P2093
P356
P1476
ORE, a eukaryotic minimal esse ...... e gene in hyperosmotic stress.
@en
P2093
Bedford JJ
Ferraris JD
García-Pérez A
Williams CK
P304
18318-18321
P356
10.1074/JBC.271.31.18318
P407
P577
1996-08-01T00:00:00Z